Developmental process of the atherosclerotic lesion.
An accumulation and swelling in artery walls that is made of macrophage cells, or debris, that contain lipids, calcium and a variable amount of fibrous connective tissue
Atherosclerosis begins as fatty streaks of the arterial wall in adolescence, progressing to hard fatty plaques that narrow and “harden” the arteries lumen in adulthood
Shock that occurs when inadequate oxygen and nutrients are supplied to the tissues because of severe left ventricular failure
A surgery where veins and arteries are used as conduit to bypass the coronary artery stenosis.
coronary artery bypass grafting (CABG)
Inflammation of the pericardium that can occur 2 to 10 weeks after a myocardial
Engorged lipid-laden macrophages that are the major component of the fatty streak
The substance that transports plasma cholesterol away from atherosclerotic plaques and to the liver for metabolism and excretion and is considered “good” cholesterol because increased levels decrease the tendency to CAD.
high-density lipoprotein (HDL)
Clenched fist over the chest is the universal sign for angina.
The main lipid component of the atherosclerotic plaque and is considered “bad” cholesterol because increased levels reflect increased tendency to CAD.
low-density lipoprotein (LDL)
Prolonged ischemia, 20 minutes or more, that results in myocardial cellular death
myocardial infarction (Ml)
Category of procedures performed during the cardiac angiography using catheters, balloons, and devices to treat atherosclerotic lesions (e.g., percutaneous transluminal coronary angioplasty [PTCA]).
percutaneous coronary interventions (PCI)
An inflammation of the pericardium
Alternating weak and strong heart beats.
Classic angina symptoms without angiographic evidence of CAD.