3611 Module II

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Author:
Anonymous
ID:
111772
Filename:
3611 Module II
Updated:
2011-10-24 13:58:20
Tags:
Endocrine
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Description:
Test two
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  1. Growth Hormone

    Targets:
    • m.
    • bone
    • soft tissue
  2. prolactin
    stimulates the production of breast milk
  3. ACTH
  4. MSH
    promote pigmentation

    target
  5. Posterior pituitary
    Vasopressin (ADH) water reabsorption

    Oxytocin- stimulates uterine contractions, ejection of breast milk
  6. Adrenal Glands
    Adrenal cortex release mineralocorticoids (regulate Na and K) and corticosteroids

    aldosterone - maintains fluid concentration

    cortisol- effects response to stress, immune function, and metabolism
  7. Adrenal Glands:

    Adrenal medulla
    Adrenal medulla release catecholamines (fight or flight)

    norepinephrine

    epinephrine
  8. Thyroid gland
    control of metabolism- T3 & T4 (increase metabolism)

    T4 converted to T3 effected by stress, starvation, beta blockers, corticosteroids,

    Table pg 1417
  9. Calcium and Phosphorus balance
    inverse relationship
  10. most common patterns affected with the endocrine system
    nutrition, activity, sexuality
  11. Hypopituitarism
    deficiency of one or more anterior pituitary hormones resulting in metabolic and sexual problems
  12. panhypopituitarism
    decreased production of all of the anterior pituitary hormones
  13. most life threatening deficiencies
    ACTH and TSH
  14. somatomedins
    enhance growth activity
  15. Hypopituitarism
    GH- short, fragile bones

    LH and FSH- decreased sexual characteristics

    ACTH- metabolism slowed

    ADH- indicates type I diabetes
  16. causes of hypopituitarism
    tumors

    anorexia

    shock

    head trauma
  17. hyperpituitarism
    occurs with pituatary tumors or hyperplasia
  18. Key features of hyperpituitarism
    tumors in cells that produce GH, PRL, ACTH

    GH-gigantism or acromegaly

    PRL-loss of 2nd degree sexual characteristics

    ACTH-cushings

    TSH- thin pt, excess energy
  19. Gigantism
    Excess in GH by anterior pituitary

    how it manifests depends on before or after puberty

    joint pain

    hyperglycemia very commno
  20. Acromegaly
    GH hypersection after puberty

    adulthood- after the closure of the epiphyses of the long bones

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