Card Set Information
bility of cell to respond to stimulus
ability of cell to reach threshold potential and generate impulses without an outside stimulus
SA node is:
pacemaker of the heart
sets pace and transmits impulses throughout the myocardium
ability of the muscle to move an impulse from cell to cell
what neurotransmitters work within the sympathetic (adrenergic) nervous system?
what do epi and norepi do in the body?
enhance force of myocardial contraction
which NT works with cholinergic parasympathetic nervous system?
--reduce force of myocardial contraction
rate of AV node?
rate of Bundle of His?
rate of bundle branches?
purkinjie fibers? 15-40
during resting state what levels are these electrolytes at INSIDE cell?
what happens to K+, Na+, and Ca++ during depolarization in reference to cells?
heart contracts due to Na+ and Ca+ moving into cell
and K+ moves out
what is action potential?
return to resting state
what is EKG?
graphic display of conduction
monophasic action potential?
depolarization and repolarization of cardiac muscle cells
what are dysrythmias?
disorders of the hearts conduction
disturbance in rate, rhythm or both
ID by analyzing EKG
5 large blocks =
contraction of the ventricles
contraction of the atrium
duration of P wave should be:
less than 3 small squares OR <.11
QRS should be:
normal PR interval?
indicates AV conduction time
steps for interpretation of EKG?
1. look for P-QRS-T
2. determine rate..atrial= count P waves, ventricular= count R waves
3. determine A-V rhythms, R-R for regularity
minor regular variations in HR and pulse pressure assoc with resp
<60 BPM, left untreated if asymptomatic
: Atropine IV, Dopamine, Epi
caused by fever, etc
Tx for atrial flutter?
anticoagulation, Cardizem, Digoxin
: aging, hypoxia, electrolyte disturbances, increased atrial pressures, tricuspid vavle disease
saw-tooth shaped waves, 200-350 threat of clotting
Tx for A fib?
: to decrease atrial irritation, decrease rate of ventricular response
: anticoagulation, Warfarin (coumadin), Digoxin, Cardizem, cardioversion if meds not effective synchronized
threat of clotting
so pt doesnt go into vfib and need defibrillation
First degree heart block?
PR = >.20 (prolonged)
delayed conduction through the AV junction
Amiodarone bolus 150 mg over 10 mins
IV flow of 1 mg/minute
may need oral antiarrythmic
Cardioversion and defib are synchronized or not?
most commonly following termination of atrial, AV junctional or ventricular tachycardias. this pause is usually insignificant
in AMI, CAD asystole may continue
: cardiac arrest
: CPR, artificial pacing, Epi, atropine
complications of pacemakers use?
dislocation of lead
sympathetic system: fight or flight:
increase HR, and BP, enhance force of contractions
during resting state, K+ is where?
V Tach Tx?
V fib tx?
epi 1 mg
vasopressin 40 U
AV node pacemaker, what type of rhythm is there?
Junctional- absent or inverted P wave
V tach can be