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so bicarb class
alkalinizing agent
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so bi p and a
raises pH of blood plasma by buffering exess hydrogen ions, vascular bicarbonate to assist the buffer system in reducing the effcts of metabolic acidosis and in the treatment of some ODs
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so bi ind
prolong crdiac arrest, known hyperkalemia, tca/barbiuate,refractory acidosis
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so bi se
extracellular alkalosis,cardiac arrhythmias,decrease cerebral and cardia functions
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so bi dose
1 mEq/kg ivp or io (1/2 dose every 10 min)
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mag sulf class
eloctrolyte, antiarrhythmic
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mag sulf p and a
acts as physiologic calcium blocker, cns depressant and anti-convulsant neuromuscular transmission, depresses the function of smooth, skeletal and cardiac muscles
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mag sulf ind
refractory v-fib, pulsless v tach, torsade de pointes, eclamptic seizure, ami
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mag sulf cont
hypocalcemia, hypertension, heart block, mycardial damageshock
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mag sulf se
aloc, hypotension, heart block, hypocalcemia
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mag sulf dose
vfib/vtach 1-2g over 1-2min, torsades de pointes 1-2 g ivp followed by .5-1g/hr, eclampti seizure 2-4 ivp/im, ami 1-2g ivp over 5 -30 min
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pralidoxime class
cholinesterase reactivator
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prax p and a
reativates cholinesterase inhibited by phosphate esters by displacing the enzyme from its recpeter sites, this restores normal functions of the neuromuscular transmission
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prax ind
an antidote and treatment for organphosphate poisoning
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pralx cont
severe cardiac disease, peptic uler, asthma, mi, hypersensitivty to procainamide/procaine
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praxl se
tc, hypertension, dizziness
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prax dose
1-2g in 250-500ml of NS infusedd over 15-30 min, no iv 1-2 g im/sq
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verapamil class
calcium channel blocker
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vera a and p
slows av conducion and suppresses reentry dysrythmias, causes coronary artery dilation and reduces o2 demand on the heart
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vera ind
SVT refractory to adenosine, afib/flutter
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vera cont
hypotension,cardiogenic shok,heart block,chf, wpw syndrome
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vera se
hypotension,heart block, chf,headache,dizziness
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vera dose
2.5-5 mg slo ivp may repeat double dose (5-10mg slow ivp) every 15-30 as needed
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amiodarone class
antidysrhythmic
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ami p and a
prolongs duration of the action potential and refractory period,relaxes smooth musles, reducing peripheral vascular resistance and increases coronary blood flow
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ami ind
treat life threatening ventricular and supraventricular dysrthmias freq a-fib
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ami cont
hypersensitivity,cardiogenic shock,bc,av blocks,liver disease
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ami se
hypotension,bc,cardiogenic shock,headache,dizziness,av block
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ami dose
vf/pulseless vt 300mg ivp/io repeat 150 3-5 min, wide complex tach 15o ivp/io over 10 min, post resuscitation maintenace drip 1mg/min ivp over 20-60 min for 6 hours max dose 2g ivp/io over 24 hours
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