cardiology PLUS medic12

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cardiology PLUS medic12
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2012-04-17 22:28:24
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cardiology medic12
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cardiology medic12 plus other things to study for final
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  1. Septal myocardial infarctions primarily involve the:
    left coronary artery
  2. The diagonal arteries of the left coronary arteries supply blood to the:
    anterilateral wall of the left ventricle
  3. Reciprocal changes that are seen in an inferior MI are most often:
    I, aVL & or v5 & v6
  4. Which two drugs can affect preload in patients with right ventricular infarctions?
    morphine & nitroglycerin only
  5. Anterior MIs most often involve which artery?
    left coronary artery
  6. ST segment elevation in the face of an anteroseptal MI would involve elevation if 1mm or more in these leads:
    v1, v2, v3, v4
  7. The condition caused by inadequate cardiac output (pump failure) is called:
    cardiogenic shock
  8. ST elevation of 1mm or more in these leads is indicative of a posterior MI:
    There are no leads facing leads for posterior MI.
  9. The leads that are reciprocal leads for a posterior MI are:
    v1, v2, v3, v4
  10. All of the following are types of EKG leads except:
    A) precordial
    B) unipolar
    C) bipolar
    D) augmented
    E) multipolar
    E) multipolar
    (this multiple choice question has been scrambled)
  11. Metabolism that occurs in the absence of O2 is termed:
    anaerobic
  12. A drug that is a naturally occurring catecholamine & stimulates both alpha & beta receptor site is:
    epinehprine
  13. Drugs that can be given down the endotracheal tube include all of the following except:
    A) valium
    B) epinephrine
    C) narcan
    D) atropine
    A) valium
    (this multiple choice question has been scrambled)
  14. The half-life of epinephrine is 3 to 4 minutes; the half life of vasopressin is:
    10 to 20 minutes
  15. The generic name for intropin is:
    dopamaine
  16. Mr. Johnson came to the hospital within the hour after his heart attack started. You would expect to see what indication of acute MI on his EKG?
    ST segment elevation in the leads over the damaged area
  17. In a lateral wall MI, which coronary artery is occluded?
    Circumflex
  18. The MI in Figure 20 is
    extensive anterior (anterior-lateral)
  19. In Figure 21 there is evidence of
    inferior MI
  20. Mrs. Campho had a previous inferior MI about 20 years ago. What would you expect to see on her EKG that would be consistent with her old inferior MI?
    significant Q wave in II, III & AVF
  21. The order in shich an MI progresses through "the tree I's" is:
    ischemia, injury, infarction
  22. AV dissociation is a hallmark of:
    third-degree AV block
  23. The rhythm in Figure 18 is
    third-degree AV block
  24. Wenchebach is characterized by
    gradually prolonging PR intervals
  25. The rhythm in Figure 10 is
    sinus rhythm w/ a sinus arrest & a ventricular escape beat
  26. Following PVCs is usually a(n)
    complete compensatory pause
  27. For which of the following rhythms is electrical shock to be the heart NOT appropriate?
    A) ventricular tachycardia
    B) torsades de pointes
    C) aystole
    D) ventricular fibrillation
    C) aystole
    (this multiple choice question has been scrambled)
  28. The rhythm in Figure 15 is
    torsades de poines
  29. Idioventricular rhythm has a heart rate of:
    20-40
  30. Multifocal PVCs are those that:
    have different shaped QRS complexes
  31. EKG RULES
    • 1) rate
    • 2) rhythm/regularity
    • 3) QRS complex
    • 4) P waves
    • 5) relationships & measurements
  32. Sinus Rhythms
    • 1) normal sinus rhythm (sinus rhythm)
    • 2) sinus bradycardia
    • 3) sinus tachycardia
    • 4) sinus arrhythmia
    • 5) sinus arrest
  33. Normal Sinus Rhythm Characteristics
    • 1) pacemaker site: SA node
    • 2) rate: 60-100 bpm
    • 3) P waves: upright in lead II, all look alike
    • 4) PR interval: generally constant; 0.12-0.20 seconds
    • 5) RR interval: usually regular
    • 6) QRS complexes: usually normal apprearing & < 0.12 seconds, may be wide
    • 7) P to QRS relationship: P precedes each QRS
  34. H's
    • 1) Hypovolemia
    • 2) Hypoxia
    • 3) Hydrogen Ion
    • 4) Hyper/Hypo-kalemia
    • 5) Hypothermia
    • 6) Hypo/Hyper-glycemia
  35. T's
    • 1) Tablets/Toxins
    • 2) Cardiac Tamponade
    • 3) Tension Pneumo
    • 4) Tamponade (AMI)
    • 5) Thromboembolism
    • 6) Trauma
  36. Right Coronary Artery Dristribution
    • 1) AV node (90% of population)
    • 2) Right Ventricle
    • 3) Inferior wall of left ventricle
    • 4) Posterior wall of left ventricle
    • (RCA forms posterior descending branch & supplies inferior wall - 90% of population)
  37. Left Coronary Artery
    • 1) Supplies bundle branches in 10% of people
    • 2) Septal Wall of left ventricle
    • 3) Anterior wall of left ventricle
    • 4) Lateral wall of left ventricle
    • 5) Posterior wall of left ventricle
  38. Lateral Wall Artery
    Left Coronary Artery
  39. Inferior Wall Artery
    Right Coronary Artery
  40. Septal Wall Artery
    Left Coronary Artery
  41. Anterior Wall Artery
    Left Coronary Artery
  42. Lead Wall's Acronym
    • L I I L I
    • S S A A L L
  43. Antidysrhythmics
    • 1) atropine sulfate
    • 2) lidocaine
    • 3) procainamide
    • 4) adenosine
    • 5) amiodarone
    • 6) verapamil
  44. Prinzmetal angina
    also known as variant angina or angina inversa, is a syndrome typically consisting of angina (cardiac chest pain) at rest that occurs in cycles. It is caused by vasospasm, a narrowing of the coronary arteries caused by contraction of the smooth muscle tissue in the vessel walls rather than directly by atherosclerosis (buildup of fatty plaque and hardening of the arteries). It occurs more in younger women.
  45. 5 Rules for a 3rd Degree Heart Block Strip
    • 1) brady
    • 2) regular P wave w/ no QRS correlation
    • 3) P wave extra but maps out
    • 4) PR interval varies
    • 5) QRS wide
  46. trycyclic OD indication & treatemnt
    1 amp Bicarb then 2 amps in 1 liter wide open (amp = 40 mEq)
  47. SLUDGE treatment
    2-5mg Atropine
  48. SLUDGE
    • Salivation
    • Lacrimation (mamary gland)
    • Urination
    • Defication
    • GI upset
    • Emisis
  49. Procainamide stop points
    • 1) up to 17mg/kg to effect
    • 2) ectopy resolves
    • 3) QRS complex widens > 50% from original
    • 4) hypotension ensues

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