Peds test #2-Endocrine

Card Set Information

Author:
sam10h
ID:
147317
Filename:
Peds test #2-Endocrine
Updated:
2012-04-12 20:45:45
Tags:
Nursing
Folders:

Description:
Peds test #2-Endocrine
Show Answers:

Home > Flashcards > Print Preview

The flashcards below were created by user sam10h on FreezingBlue Flashcards. What would you like to do?


  1. Incidence of 1 in 4000; Screening measures levels of thryoid simulating hormone
    Congenital Hypothyroidism
  2. Incidence 1 in 13,000; deficiency in phenylalanine hyroxylase
    Phenylketonuria
  3. S/S of congenital hypothyroidism
    Poor feeding, lethargy
  4. S/S of PKU
    vomiting, irritability, hyperactivity
  5. incidence of 1 in 30,000; absencse of galactose-phosphate uridyl transferase; S/S cataracts, weight loss; treat with lactase formula
    Galactosemia
  6. PKU must be tested after ingestion of what?
    Protein
  7. What signifies a positive Guthrie test for PKU?
    >4mg/dl
  8. Treatment for PKU includes:
    Restrict dietary protein, keep phenylalanine in a safe range (2-8 mg/dl); begin diet before 3 weeks of age; maintain low protein diet for life
  9. What is the master gland that is responsible fo regulating other glands?
    Pituitary gland
  10. The anterior pituitary gland produces what hormones?
    GH, sex hormones, TSH
  11. The posterior pituitary gland produces what hormones?
    ADH, oxytocin
  12. Pituitary gland is controlled by hormones that are produced by the ___
    hypothalamus
  13. Inhibits somatic growth; primary site of dysfunction is hypothalamus; most common causes is pituitary tumors/hypothalamic tumors
    Growth Hormone Deficiency
  14. What are some clinical manifestations of GH deficiency?
    Appears younger than age; bone age is retarded; permanent teeth are delayed; sexual development is delayed
  15. Daily treatment of ___ to ___ ug/kg of biosynthetic growth hormones is needed in GH deficiency until acceptable height is achieved
    25 to 50
  16. Nursing considerations for hormone replacement therapy for GH deficiency includes: (3)
    administer at bedtime; administer 6 of 7 days; encourage self management in school age children
  17. Excessive growth hormone before closure of the epiphyseal shafts results in:
    overgrowth of long bones
  18. excess GH after epiphyseal closure is ___
    acromegaly
  19. Typical facial features of acromegaly
    Separation, malocclusion of teeth, prominent forehead, increased facial hair
  20. Therapeutic management of acromegaly (3)
    Surgery to remove tumor, radiation, hormone replacement therapy after surgery
  21. Nursing considerations for acromegaly (4)
    identify children with excessive growth rates early; early treatment; emotional support; body image concerns
  22. Sexual developments before age 9 in boys/8 in girls
    Precocious Puberty
  23. Isolated appearance of breast development
    Premature thelarche
  24. Early appearance of pubic hair
    Premature pubarche
  25. What does the medication Lupron treat?
    Peripheral Precocious Pseudopuberty; slows prepubertal growth to normal rates; discontinue at puberty!!
  26. the principal disorder of the posterior pituitary
    Diabetes Insipidus (DI)
  27. Results from hyposecretion of ADH; produces uncontrolled diuresis
    DI
  28. Secondary causes of DI includes (4)
    trauma, tumors, CNS infection, aneurysm
  29. Some clinical manifestations of DI include: (7)
    Polyuria, polydipsia, enuresis (usually 1st sign), irritability, dehydration, hypernatremia, sometimes fever
  30. DI needs a daily hormone replacement of ___
    Vasopressin
  31. Drug of choice for DI
    DDAVP
  32. Nursing considerations for DI: (5)
    Identify disorder, I&O, observe for s/s of fluid overload, seizure precautions, administer ADH antagonizing meds
  33. SIADH stands for:
    Syndrome of Inappropriate Antidiuretic Hormone
  34. produced by hypersecretion of the posterior pituitary (increased ADH)
    SIADH
  35. S/S of SIADH (5)
    Fluid retention, hypotonicity, decreased sodium, anorexia, iriitability, personality changes
  36. Treatment of SIADH (3)
    Fluid restriction, Seizure precautions, long term ADH antagonizing medications
  37. Thyroid hormone regulates what?
    BMR
  38. Thyroid hormone secretes what 2 hormones?
    Thryoid hormone (T4/T3) and thyrocalcitonin (which maintains blood calcium levels)
  39. What hormone stimulates the thyroid?
    TSH
  40. Most commonly results from agenesis, dysplasia, ectopy of thyroid; can also becaused by autosomal recessive defects
    Juvenile Hypothyroidism
  41. Clinical Manifestations of Juvenile Hypothyroidism (9)
    Decelerated growth, constipation, sleepiness, skin changes, elevated weight, bradycardia, large fontanelles/tongue, delayed dentition, goiter development
  42. Hypertrophy of the thyroid gland
    Goiter
  43. (Congenital) usually results from maternal ingestion of antithyroid drugs during pregnancy
    Goiter
  44. (acquired) result of neoplasm, inflammatory disease, dietary deficiency, increased secretion of pititary thyrotropic hormone
    Goiter
  45. Therapeutic management for goiter (4)
    oral thyroid hormone replacement, prompt treatment for brain growth in infant, administer in increasing amounts over 4-8 weeks, compliance
  46. Nursing considerations for Goiter (4)
    Thyroid enlargement may compromise airway in infant; may be noticable during periods of rapid growth; large goiters may be obvious upon palpation; TH replacement is necessary!
  47. most common cuase of hyperthyroidism in childhood is what?
    Graves disease
  48. Peak incidence of hyperthyroidism is between ___ and ___ years old
    12 to 14
  49. Some clinical manifestations of Grave's disease: (8)
    Exophthalmos, weight loss, increased appetite, nocturia, palpitations, irritability, sleeplessness, inability to concentrate
  50. PTU specifically inhibits the peripheral conversion of ___ to ___
    T4 to T3
  51. Transmitted as autosomal dominant disorder with formation of structurally abnormal insulin with decreased biologic activity
    Maturity Onset Diabetes of the Young
  52. The first symptom of DM is usually:
    Nocturnal enuresis
  53. S/S of hypoglycemia (3)
    Dizziness, confused, shaky
  54. acute metabolic changes in the body due to lack of insulin; dehydration, acidosis, hyperglycemia
    Ketoacidosis
  55. Hyperventilation characteristic of metabolic acidosis resulting from respiratory system's attempt to eliminate CO2 (in DKA)
    Kussaul Respirations
  56. Rapid acting insulin; what is the onset, peak, and duration?
    Insulin lispro (Humalog); onset is 10 to 30 mins, peak is 1-2 hours, duration 2-4 hours; give 15 mins before/after a meal
  57. Short acting insulin; onset, peak, duration?
    Regular insulin (Humulin R, Novolin R); onset 0.25 to 1 hr; peak is 1.5 to 4 h; duration 5 to 9 hr
  58. Intermediate acting insulin; onset, peak, duration?
    NPH (Humulin N, Novolin N); onset 3-4 hrs; peak 8-14 hrs; duration 16-24 hrs
  59. The adrenal cortex secretes 3 groups of what steriods?
    Glucocorticoids (cortisol, corticosterone); Mineralocorticoids (aldosterone); sex steroids (androgens, estrogens, progestins)
  60. Adrenal medulla secretes what? (2)
    Epinephrine, norepinephrine
  61. What kind of tumors are the primary cause of adrenal medullary hyperfunction?
    Catecholamine-secreting tumors
  62. "Adrenal crisis"; caused by hemorrhage from trauma, fulminating infections, abrupt withdrawal from exogenous cortisone, failure to increase cortisone in times of stress
    Acute adrenocortical insufficiency
  63. Symptoms of Acute adrenocirtical insufficiency in infants includes: (4)
    High temp, tachypnea, cyanosis, seizures
  64. S/s of acute adrenocoritcal insufficiency in children: (5)
    irritability, headache, abdominal pain, weakness, diarrhea
  65. nursing considerations for acute adrenocortical insufficiency (6)
    Recognize symptoms promptly, monitor vital signs, seizure precautions, monitor fluid therapy/electrolytes, support parents
  66. Rare in children; usually occurs as a result of neoplasm, lesion of adrenal glands, idiopathic cuase
    chronic adrenocortical insufficiency
  67. Side effects of this medication are gastric irritation, irritability, sleeplessness, weight gain, depression, euphoria
    Glucocorticoids (medication for chronic adrenocortical insufficiency)
  68. Side effects of this medication includes generalized edema, hypertension, headache, cardiac arrhythmias, hypokalemia
    Mineralcorticoids (used for chronic adrenocortical insufficiency)
  69. Characteristic group of manifestations caused by excessive circulating free cortisol; may be caused by excessive/prolonged steroid therapy
    Cushing syndrome
  70. A person with this appearance has excessive hair growth, moon face, red checks, weight gain, pendulous abdomen with red striae, poor wound healing, ecchymoses
    Cushingoid appearance
  71. Post op complications of Cushing syndrome: (3)
    anorexia, muscle/joint pain, depression
  72. Therapeutic management of cushing syndrome (2)
    Bilateral adrenalectomy, replacement of GH, ADH, TH, gonadotropins, steroids

What would you like to do?

Home > Flashcards > Print Preview