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This slows conduction through AV node, interrupts AV nodal reentry pathways & restores NSR in PSVT. Half life is < 10sec due to red blood cell sequestering
Sympathomimetic that is relatively selective for Beta 2 adrenergic receptors.
Bronchodilator with few cardiovascular effects
Duration 4-6 hrs
Class IIb Antidysrhythmic for ventricular dysrhythmias.
Prolongs action potential duration in all cardiac tissues
NSAID that inhibits platelet Aggregation
Enhances sinus node automatacity and AV conduction via vagolytic action
Dextrose is fuel for cellular metabolism and is regulated by insulin
Acts as a tranquilizer, anticonvulsant, and skeletal muscle relaxant (via CNS)
Reverses effects of phenothiazines and has anticholinergic and antiemetic effects
Beta 1 receptor with positive inotropic and negative chronotropic effects
- inotropic = muscle force
- chronotropic = heart rate
Beta stimulator with positive chronotropic and inotropic effects.
Relaxes smooth muscle of the bronchi and is therefore effective in treating bronchoconstriction associated with asthma and anaphylaxis
Increases blood flow to heart during CPR
50-100 times more potent than Morphine with shorter duration of action. Principal therapeutic values are analgesic and sedative
Resp rate and alveolar ventilation changes might last longer than analgesic effect.
Onset is immediate
Inhibits reabsorbtion of sodium and chloride in kidney, producing peak diuresis. Rapidly reduces extracellular fluid, decreases venous return and ventricular output by causing venous dilation (3-4min).
Sympathomimetic that is selective for Beta 2 adrenergic receptors.
Causes relaxation of bronchial smooth muscle, thus decreases airway resistance and increases vital capacity.
Greater affinity for B2 adrenergic receptors than its chemical variant
Increases Fibrillation threshold of myocardium and decreases automatacity, supressing ventricular ectopy.
Decreases stimulation of CNS during intubation
Has selective Beta 1 adrenergic receptors.
Decreases HR, systolic BP, and Cardiac output
Effective in reducing incidence of V-Fib & Chest pain in MI patients.
Acts as a amnesic, hypnotic, antiolytoc, and anticonvlsant
3-4 times more potent than other benzodiazepine
Reduces anxiety associated with pain (important in cardiac related pain)
CNS depressant, increases peripheral venous capacity
Decreases venous return, decreases myocardial demand
Narcotic antagonist that binds with narcotic receptor sites
Nasal decongestant that causes vasoconstriction of superficial blood vessels in nasal mucosa
Smooth muscle relaxant causing vasodilation.
Decreases cardiac preload.
May improve perfusion t ischemic myocardium by dilating coronary arteries.
May provide a significant degree of pain control in patients with cardiac chest pain
Reduces the workload on the hear and lungs to maintain proper perfusion.
Facilitates breakdown of glucose into a usable energy for to maintain aerobic metabolism
Alkalizing agent that binds with excess ions to correct metabolic acidosis
Synthetic adrenal corticosteroid used as an antiinflammatory in spinal injury and in the management of acute and chronic inflammatory diseases.
Inhibits inflammatory response by interfering with many of the substances that cause inflammation (cytokines, interleukin, interferon)
This is required for the conversion of pyruvic acid to acetyl coenzyme A.
With out this a significant amount of energy available in glucose cannot be obtained.
- Thiamine B1
- Any comatose patient (especially those suspected to be alcoholic) should receive thiamine in addition to D50 or Narcan
A naturally occuring anti-diuretic hormone which becomes a powerful vasoconstrictor when used at a higher dose than normal.
Exerts a greater vasoconstrictive effect under conditions of hypoxia and acidosis than its co-drug. It also lasts longer, has greater arterial tone, and therefore, has an effect that correlates with greater myocardial perfusion.
It does not increase myocardial oxygen consumption and lactate production in the arrested heart.
This has mild sedative properties, is tolerated well in pediatric populations, is safe in pregnancy, and does not produce extrapyramidal effects of phenothiazines.
Serotonin 5-HT3 receptor antagonist antiemetic
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