Chapter 21, APSGN

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tville01
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155804
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Chapter 21, APSGN
Updated:
2012-05-25 07:52:32
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Chapter 21 APSGN
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Chapter 21, APSGN
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  1. degenerative inflammation of the glomeruli
    acute poststreptococcal glomerulonephritis(APSGN)
  2. What is the causitive agent for APSGN
    group A beta-hemolytic streptococcus
  3. What is APSGN associated with
    streptococcal infection
  4. Describe the patho of APSGN
    • antibodies created for streptococcus
    • antigen/antibody complexes formed
    • complexes deposit to basement membrane of glomeruli
    • activates compliment
    • innflammatory response, WBC arrive
    • cappillary permability increases
    • glomeruli swell,filtration decreased,urine decreased
    • renin released, trigger aldosterone
    • absorps sodium & water
    • leads to edema and high BP
  5. What are the signs of APSGN
    • flank or back pain-swelling kidneys
    • hematuria
    • RBC casts
    • proteinuria/albuminuria
    • hypoalbuminemai=edema
    • secondary hypertension-renin & reduced GFR from inflammed glomeruli
    • olguria
    • urine dark and cloudy-protein & RBC in urine
  6. What are the diagnostic test for APSGN
    • history
    • urinalysis-proteinuria,gross hematuria,RBC cast
    • BUN & serum creatine elevated-decreased GFR
    • elevation of streptococcal enzymes-ASO ASK
    • acidosis
    • renal biopsy
    • serum complement decreased
  7. What are the complications of APSGN
    • kidney scarring
    • acute renal failure
    • chronic glomerulornephritis
  8. what is the treatment for APSGN
    • sodium restrictions
    • restrictions in protein and fluid
    • steroids
    • antihypertensives
  9. what is the prognosis for APSGN
    good

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