Perio c_5 & 6.txt

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Author:
tigermom23
ID:
17993
Filename:
Perio c_5 & 6.txt
Updated:
2010-05-05 19:24:10
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perio chpt
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perio chapt.5 and 6
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  1. Calculus removal plays a bigger part in perio disease than plaque removal
    False
  2. Etiologic cause of perio disease
    Plaque
  3. Can plaque grow on top of calcus
    Yes
  4. Calculus is formed by -_____in bacterial plaque
    Calcium and phosphate salts
  5. Calculus above the gumline
    Supragingival
  6. Calculus below the gumline
    Subgingival
  7. What was thought of as the cause of perio diseas
    calcium and phosphafe salt
  8. What is Pellicle formed from?
    Salavary glycoproteins
  9. Plaque grows on top of the calculus and therefore the bacteria is close to the tissue and prevents proper plaque control
    True
  10. Why is bone loss increased in areas with amalgam overhangs
    A source of plaque retention
  11. What is the anti calculus agent
    Pyrophosphate
  12. How do we measure the severity of periodontal disease
    Clinical attachment loss
  13. Other factors that contribute to perio disease
    • Orfho disease
    • Mal occlusion
    • Mouth breathing
    • Tobacco use
  14. What initiates gingivitis
    Plaque
  15. Light calculus formers have high levels of....
    Parotid pyrophosphate
  16. What is a sign of someone who is a "mouth breather"
    Gingival tissue of the maxillary anterior is red and irritated
  17. Is there a cure for mouth breathing
    No- treatment is only palliative
  18. "redness"
    Erithemia
  19. Inflammation of the gingival tissue without bone loss or attachment loss
    Gingivitis
  20. What are some changes in the tissue when gingivitis is present
    • Redness
    • Edema(swelling)
    • Exudate(drainage)
    • Bleeding
  21. What reason is gingivitis often unrecognized?
    It is painless
  22. Why does probing depth increase in gingivitis
    Tissue swells
  23. Initial stage of gingivitis - acute response
    Stage I
  24. Order of stage I gingivitis
    • Vasoconstriction
    • Vasodilation
    • Margination
    • Migration
  25. What stage of gingivitis has the first clinical signs
    Stage II
  26. Common signs of class II gingivitis
    • White or yellow exudate
    • Red/swollen gjngivitis
    • Bleeding on probing
  27. Day 4-7 gingivitis
    Stage II
  28. Established stage of gingivitis
    Stage III
  29. Signs of stage III gingivitis
    • Destroyed collagen (spongey)
    • Pocket epithelium forms
    • Probe readings increase
    • Junctiional epithelium permeable
    • Edema of gingival margin
  30. Which stage may persist for years / months OR reversed
    Stage III
  31. Grows in disease and causes pocket epithelium-not present in health
    Rete pegs
  32. Known as the advanced stage-begins to extend into the bone
    • Stage IV
    • (preriodontal disease)
  33. Healing after gingiva treatment occurs in what order
    • Reverse process
    • Begins @ connective tissue and finally epithelium
  34. The most common form of gingivitis
    Gingivitis associated wirh plaque
  35. Signs of gingivitis associated with plaque
    • Ulceration of tissue
    • BOP
  36. Over growth of gingiva tissue
    Hyperplastic
  37. Systemic factors intensify gingival response to plaque
    True
  38. Type of meds that causes hyperplasia
    • Phenytoin/dilantin
    • Verapamil / nifedipine
    • Cyclosporine
  39. Phenytoin / Dilantin
    Seizure med
  40. Verapamil / Nifedipine
    Cardiac
  41. Cyclosporine
    • Immunosupperssion
    • (organ transplant)
  42. Deficiency of this vitamin causes defects in collagen
    Vit C
  43. Necrotizing Ulcerative Gingivitis
    NUG
  44. Symptoms of NUG
    • Rapid onset
    • Ulcerative tissue
    • Punched out pipillae
    • Pseudomembrane
    • Fetor oris
  45. Bacteria that is responsible for NUG
    • P.Intermedia
    • Spirochete(Treponema Denticola)
  46. In younger children
    Elevated temp
    Vesicles formation
    No fetoris Oris
    Gingival herpetic gingivostomatitis
  47. Yeast organisms that cause candidis
    Candida albicans
  48. How is fungal origin gingivitis treated
    Antifungal medication
  49. LGE-found in HIV patients
    Linear gingival erythema
  50. Contains white patches that will rub off
    LGE
  51. Gingival enlargement due to the # of cells
    Hyperplasia
  52. Gingival enlargement due
    To cell size
    Hyperyrophy
  53. Hemorrhagic and swollen gingiva
    Acute leukemia
  54. Lacy white lines - caused by systemic conditions
    Wickham's striae
  55. Chronic - immune related caused by stress
    Lichen Planus
  56. Blistering and sloughing of the gingival epithelium-Nikolsky's sign
    Mucous Membrane Pemphigoid
  57. Pemphigoid leasions-painful and raw-due to allergic reactions
    Desquamative Gingivitis
  58. 2 OTHER NAMES FOR SUBGINGIVAL CALCULUS
    • SUBMARGINAL CALCULUS
    • SERUMAL CALCULUS
  59. ONE OF THE MOST COMMON TYPE OF PLAQUE TRAP
    AMALGAM OVERHANG
  60. WHAT LEADS TO LOCALIZED GINGIVAL INFLAMMATION--CONFINED TO THE LABIAL GINGIVA OF THE MAXILLARY ANTERIOR TEETH
    MOUTH BREATHING
  61. STUDIES SHOW THAT SMOKING IS ASSOCIATED WITH DEEPER POCKETS AND MORE CLINICAL ATTACHMENT LOSS
    TRUE
  62. THE TYPE OF CALCULUS THAT IS TIGHTLY ADHERENT TO THE TOOTH AND FOUND NEAR OPENINGS OF THE MAJOR SALIVARY GLANDS
    SUPRAGINGIVAL
  63. STEP ONE TO CALCULUS FORMATION
    FORMATION OF PELLICLE
  64. ANTICALCULUS TOOTHPASTES REDUCE THE FORMATION OF NEW SUPRAGINGIVAL CALCULUS BY HOW MUCH?
    20% TO 30%
  65. WHAT PERCENT OF THE POPULATION OF DENTAL PATIENTS HAVE AMALGAM OVERHANGS
    UP TO 75%
  66. STAGES OF GINGIVITIS
    • STAGE I - INITIAL OR SUBCLINICAL STAGE
    • STAGE ii - EARLY STAGE
    • STAGE III - ESTABLISHED STAGE
    • STAGE IV - ADVANCED STAGE
  67. REFERS TO THE LOCATION OF THE MARGIN OF THE TISSUE - NOT THE CONDITIONS
    RECESSION
  68. FETIS ORIS
    ODOR CAUSE BY NUG
  69. NO CURE - ONLY TREATING SYMPTOMS
    PALLIATIVE
  70. TISSUE DAMAGE CAUSED BY TRAUMA
    TRAUMATIC LESIONS
  71. WHEN CICATRICAL PEMPHIGOID LESIONS ARE LIMITED TO THE GINGIVA
    DESQUAMATIVE GINGIVITIS
  72. ETIOLOGY OF DESQUAMATIVE GINGIVITIS
    AUTOIMMUNE OR ALLERGGIC REACTTIONS
  73. IMMUNE RELATED, AFFECTS THE SKIN AND MUCOUS MEMBRANES OF MIDDLE AGED PATIENTS
    LICHEN PLANUS
  74. REACTION TO A FOREIGN BODY IN THE TISSUE
    FOREIGN BODY REACTIONS
  75. SWELLING
    EDEMA
  76. WHICH STAGE OF GINGIVAL INFLAMMATION BEGINS WITH IN 4-7DAYS AFTER PLAQUE HAS ACCUMULATED - MAY BE SEEN AS SLIGHTLY RED AND SWOLLEN
    STAGE I - INITIAL STAGE (NO CLINICAL SIGNS)
  77. STAGE OF GINGIVAL INFLAMMATION CAN BLEEDING FROM THE SULCUS BE DETECTED WITH A PERIODONTAL PROBE
    STAGE II - EARLY STAGE
  78. WHY DOES CLINICAL PROBING DEPTH INCREASE IN STAGE III GINGIVITIS
    TISSUE BECOMES ENGORGED WITH INFLAMMATORY CELLS

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