FMS3 Path

Card Set Information

Author:
NicoleCoppage
ID:
180013
Filename:
FMS3 Path
Updated:
2012-10-26 01:12:55
Tags:
Path
Folders:

Description:
Abnormal Bloody Supply I
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  1. A) Transudate


    B) Exudate
    • A) Non-infmalitory
    •      ^ Hydrostatic Preassure
    •     Decrease Onconic P
    •     No change to vascular Perm

    B) Inflamitory/ ^ vascular Perm/direct Vasc dmg
  2. Hyperemia
    Active!

    • Arteriol Vasodil= ^ blood flow
    • Red tissue/warmer
  3. Congestion
    Passive

    • Imparied outflow
    • Cyanotic tissues
  4. Pulmonary edema
    A) Acute
    B) Chronic
    Congestion

    A) Sudden Lv failure- ^ preasure- engorged capilary beds = transudate creation. Frothy pink edema thats soffocates

    B) Persistent, less pressure. Aveolar walls respond by thickining. ^ macrophages heart failure cells
  5. Liver Congestion
    A) Acute
    B) Chronic
    Congestion

    • A) 2ndry to shock/hepatic vein thrombosis. Central vein/centrilobar hepatocytes distended w/ blood.
    • Blood flows from Portal tract to central vein- centrilobar hepatocyts vulerable to hypoxic injury

    B) RV failure. Centrilobar hepatocyt necrosis. Nutmeg liver
  6. Sequence of Hemostatsis
    • 1) Exsposure to subendothelial collagen leads to platlet binding and activation
    • 2) Platlets adhere and aggregate at injury site= primary platlet plug
    • 3) @ same time, Activation Coag Casscade
    • 4) Prothrombin converted to active form (thrombin) --> catalyses circulating fibrinogen to fibrin--> platlet plug
  7. Hemorrrhage
    A) small vessel
    B) large vessel
    A) 2ndry to congestion & ^ preasure

    B) Trauma/Atherosclerosis etc

    * Excessive hemorage = jaundice due to release of bilirubin following redblood cell breakdown
  8. Virchow Triad
    • 1) Endothelial injury
    • 2) Stasis or turbulent blood flow
    • 3) blood hypercoaguability
  9. Endothilial injury risk factors
    MI/ hypertension/ hypoxia/ cigarette smoke/ Bacterial endotoxin/ radiation/ trauma-sugury/vasculitus
  10. Blood Turbulence
    Cells/platlets in center, plasma normaly flow along vessel phriphery. Disruption of laminar flow increases chance of contact between platlets and vessel wall.
  11. Arterial Thrombosis
    Result of Atherosclerotic vessel disease

    turbulance arises at branch points- Aortic/iliac/carotid artery bifurcations

    Common sites: Coronary/cerebral/femoral arteries.

    More compact and pale than Venious. More platlets/fibrin/leukocytes. less RBC

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