Cardiology II.txt

Card Set Information

Author:
Anonymous
ID:
181244
Filename:
Cardiology II.txt
Updated:
2012-11-01 22:15:49
Tags:
Positive Inotropic
Folders:

Description:
Cardiovascular System Drugs
Show Answers:

Home > Flashcards > Print Preview

The flashcards below were created by user Anonymous on FreezingBlue Flashcards. What would you like to do?


  1. SubClass: Cardiac Glycosides
    Prototype: digoxin (Lanoxin)

    ACTION
    • Action:
    • +Inotropic - Inhibits the sodium thus increases Na+ and Ca+. Increases cardiac oitput and increases contraction.
    • -Dromotropic - Decreases the rate of conduction velocity. Prolongs refactory period.
    • -Chronotropic - Decreases HR, and increases filling time.
    • Increases Stroke Volume -
  2. SubClass: Cardiac Glycosides
    Prototype: digoxin (Lanoxin)

    INDICATIONS
    Indications: CHF, Atrial Fibrillation, Atrial Flutter
  3. SubClass: Cardiac Glycosides
    Prototype: digoxin (Lanoxin)

    SIDE EFFECTS
    • Side Effects: Has a very narrow theraputic window
    • Decreases K+, Ca+, leavels = increased toxicity
    • GI: anorexia, nausea, vomiting, diarrhea
    • Visual: Green/yellow halos
    • CNS: confusion, fatigue
    • CV: Bradycardia, Heart Block, disrhythmias
  4. SubClass: Cardiac Glycosides
    Prototype: digoxin (Lanoxin)

    NSG IMPLICATIONS
    • Nsg Implications: takes 7 days to reach theriputic levels
    • star with loading dose
    • typical oral dose is 0.125 or .25 mg daily
    • IV push over 5 min
    • dig levels noramal is .5 to 2
    • toxicity occures in as many as 20%
    • Diet: No large amounts of brain, antacids and milk products should not be taken within 2 hours of dose
    • Tape dose as needed

  5. SubClass: Cardiac Glycosides
    Prototype: digoxin (Lanoxin)

    TOXICITY
    • Toxicity: S/S Cardiac Arrhythmia, anoxeria, nausea, vomiting, weakness, fatigue, green/yellow halos in vision
    • TX: stop digoxin, treat arrhythmia, monitor leavels and EKG,
    • Andidote: digibind and increase K+
  6. SubClass: Phosphodiesterase inhibitors
    Prototype: milrinone (Primacor)

    ACTION
    • Action:
    •      + Inotropic: Inhibits phosphodiesterase type III (enzyme) founs in heart and smooth muscle thus - increases CAMP and increases Ca+
    •      Vasodilation: Decreases work load on heart
    •      +Lusitropic : relaxation of blood vessels
    •      +Chronotropic
  7. SubClass: Phosphodiesterase inhibitors
    Prototype: milrinone (Primacor)

    INDICATIONS
    Indications: Short term management of CHF, for those who don't respond to digoxin, diuretics, vasodilators
  8. SubClass: Phosphodiesterase inhibitors
    Prototype: milrinone (Primacor)

    SIDE EFFECTS
    Side Effects: Ventricular dysrhythmia, Hypotension, angina, hypokalemia, tremor, thrombocytopenia
  9. SubClass: Phosphodiesterase inhibitors
    Prototype: milrinone (Primacor)

    NSG IMPLICATIONS
    Nsg Implications: Furosemide is contraindicated, infuse with pump ONLY, Continualy monitor EKG
  10. SubClass: Phosphodiesterase inhibitors
    Prototype: milrinone (Primacor)

    TOXICITY

    • Toxicity: NO ANTIDOTE
    • Hypotension most common toxic effect

    D/C med, provide support Tx

What would you like to do?

Home > Flashcards > Print Preview