PTG 105- Exam 3- Lecture 20 - 3

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kyleannkelsey
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PTG 105- Exam 3- Lecture 20 - 3
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2013-04-09 22:58:22
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PTG 105 Exam Lecture 20
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PTG 105- Exam 3- Lecture 20 - 3
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  1. What is Osteitis fibrosa cystica?
    Bone converted to fibrous tissue with cysts
  2. What is the name for bone converted to fibrous tissue with cysts?
    Osteitis fibrous cystica
  3. Parathyroid adenoma is the most common cause of what?
    hyperparathyroidism
  4. What is the most common cause of hyperparathyroidism?
    Parathyroid adenoma
  5. How do you treat parimary hyperparathyroidism?
    Surgical removal
  6. How do you treat secondary hyperparathyroidism?
    treat the primary disease
  7. What are the causes of Hypoparathyroidism?
    • surgical removal of parathyroid due to hyperparathyroidism
    • removal during thyroidectomy
    • congenital absense
    • autoimmune
    • idiopathic
  8. What is DiGeorge's syndrome?
    Congenital absense of parathyroid gland
  9. What is the name for the congenital absense of the parathyroid gland?
    Digeorge syndrome
  10. What reasons would you surgically remove teh parathyroid?
    • incidental due to removal of thyroid
    • Hyperparathyroidism
  11. How does Hpyoparathyroidism manifest itself?
    Decreased serum Ca (causes muscle spasms and convulsions)
  12. A person with decreased Ca in the serum and muscle spasms and convulsions might have what disease?
    Hypoparathyroidism
  13. How do you test for Hypoparathyroidism?
    Perform lab testing for decreased PTH and Ca
  14. What is the treatment for hyperparathyroidism?
    Synthetuc Vitamin D and Ca supplements
  15. What disease can be treated with synthetic Vit. D supplements and Ca supplements?
    Hypoparathyroidism
  16. What three general groups of hormones are produces by the Adrenal cortex?
    • Mineralcorticoids
    • Glucocorticoids
    • Weak androgens
  17. What general group of hormones are produced in the adrenal medulla?
    Catecholamines
  18. What is the purpose of mineralcorticoids?
    Na and K balance
  19. What is the purpose of Glucocorticoids?
    • raise blood glucose
    • inhibit inflammation
    • adapt to long term stress
  20. WHat do catecholamines do?
    Increae blood pressure HR and deal with short term stress
  21. Zona glomerulaosa produces what general hormones?
    Mineralcorticoids
  22. Zona fasciculata produce what general types of hormones?
    Sex steroid and glucocorticoids
  23. Zona reticularis produces what types of hormones?
    Glucocorticoids and sex steroids
  24. Name four diseases of the adrenal glands
    • Addison's disease
    • Cuching's disease
    • Conn's disease
    • Neoplasm of adrenal medulla
  25. What does Addison's disease, Chuching's syndrome, Conn's syndrome all have in common?
    They are diseases of the adrenal glands
  26. What is Addison's disease?
    insuffiecnt production of adrenocorticoids
  27. What disease is characterized by insufficeint production of adrenocorticoids?
    Addison's disease
  28. What is cushing's disease?
    Excess corticosteroids, particularly glucocorticoids
  29. What disease is characterized by excess production of corticosteroids, paricularly glucocorticoids (cortisol)?
    Cuching's syndrome
  30. Conn's disease is:
    Overproduction of mineralcorticoids
  31. What disease is characterized by the overproduction of mineralcorticoids (aldosterone)?
    Conn's syndrome
  32. What is the main glucocorticoid?
    cortisol
  33. What is the main mineralcorticoid?
    aldosterone
  34. What neoplasm of the adrenal medulla is found in children?
    Neuroblastoma
  35. What neoplasm of the adrenal medulla is found in adults?
    Pheochromocytoma
  36. What is characteristic of neuroblastoma and pheochromocytoma?
    They both have excess production of catecholamines
  37. If an adult has excess production of catecholamines, what disease is suspect?
    Pheochromocytoma
  38. If an child has excess production of catecholamines, what disease is suspect?
    Neuroblastoma
  39. What causes Addison's disease?
    tuberculosis and idiopathic adrenal atrophy
  40. How does Addison's disease manifest itself?
    • Increased skin pigment
    • Tiredness
    • Intestinal complaints
    • Hypotension
    • Hypoglycemia
    • Loss of body hair
    • depression
    • decreased serum corticosteroids
    • Increased ACTH
  41. If person presnets with decreased body hair, depression, intestinal upset, increased pigmentation on skin,  tiredness, decreased serum corticosteroids and increased ACTH, what would the diagnosis be?
    Addison's disease
  42. Where do skin pigmentation changes happen in aperson with Addison's disease?
    skin folds and pressure points, such as knees, elbows, knuckles, toes, mucous membranes
  43. Acute addison's disease needs what treatment?
    • life threatening:
    • immediate treatment with glucocorticoids and IV fluids with salt and sugar
  44. What is the treatment for Chronic Addison's disease?
    Corticosteroids
  45. What cushing's syndrome is caused by what four things?
    • adrenal cortical tumor
    • tumor in anterior pituitary
    • lung tumror
    • Steroids

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