Pharmacology: Cardiac meds

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FireJosh
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219160
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Pharmacology: Cardiac meds
Updated:
2013-05-11 23:30:29
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mixture of questions from book and lecture outline
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  1. Arrythmias are caused by either abnormal _____ or abnormal ___________ blank conduction
    • automaticity
    • impulse
  2. Antiarrythmics directly or indirectly alter the abiity of _______ to pass through cell membranes suppressing abnormal automaticity, changing the refractory period in myocytes , or slowing conduction
    ions
  3. Class 1a increase the effective ________ period through sodium channel blockade
    refractory
  4. Procainamide class
    1a
  5. lidocaine class
    1b
  6. lidocaine shortens the refractory period and action potential cycle which reduces tendancy to depolarize in __________ fasion
    disorganized
  7. Lidocaine has greater impact on________cells with longer action potential than on _______ cells with shorter action potential.
    • ventricle
    • atrial
  8. Lidocaine has largely been replaced by _______
    reaches peak effect in 5 minutes but has a variable duration of
    • amiodarone
    • action
  9. flecainamide is class _________. is given  ________ (route) and has an increase in mortality (rarely used)
    • 1c
    • oral
  10. Beta adrenergic antagonists are class. They inhibit Ca and K influx by blocking stimulation from _______ and ___________ in pacemaker cells
    • 2
    • epinephrine and norepinephrine
  11. amiodarone is a class
    3
  12. class III blocks ________ channels
    potassium
  13. myocardial cells depolarize through _____influx and repolarize through  _______ efflux. In contrast pacemaker cells depolarize by an influx of __________ and repolarize through ________ efflux
    • Na
    • K
    • Ca
    • K
  14. Ca channel blockers are class _____. The two drugs in this classification are _______ and ______
    • IV
    • verapamil
    • diltiazem
  15. Ca channel blockers decrease conduction velocity through _____ node. and are used to treat _______ tachycardias
    • AV
    • atrial
  16. class V is composed of miscellaneous agents including ________ and __________
    • digoxin 
    • adenosine
  17. Lidocaine is less effective than _______ in monomorphic v tach
    procainamide
  18. _________ posesses electrophysiologic characteristics of all 4 classes and is indicated for: a fib, refractory SVT's, V arrythmias,
    amiodarone
  19. Adenosine slows conduction and prolongs refractory period in ______ node
    AV
  20. ____________has a half live of 9 seconds
    Adenosine
  21. Anticoagulant do not __________ existing clots
    dissolve
  22. Warfarin interferes with hepatic synthesis of _____ dependant coagulation factors (II VII IX X)
    vit k
  23. Warfarin has multiple profound food and medication ____________
    Interactions
  24. Heparin activates antithrombin III and inactivates ______ _________ most importantly thrombin
    coagulation factors
  25. Protamine is the ________ for heparin and is used in OD of low molecular weight heparins
    antagonist
  26. _______ is the antagonist for supra theraputic INR with warfarin
    vit k

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