Blood parasites

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ucstudent01
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246528
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Blood parasites
Updated:
2013-11-12 12:13:39
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Blood Parasites
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blood parasites
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  1. Plasmodium species
    • sprozoites invade liver
    • Merozoites invade RBC'S
    • Gametocytes infect mosquito
  2. P. vivax
    • Large RBC's
    • Troph irrecgular
    • Multi-phases seen, gametocytes appear early
    • could see baso stip.
  3. P. malariae
    • Normal RBC's
    • troph compact - Band Forms may be seen
    • Gametocytes seen after weeks
  4. P. falciparum
    • Most common-
    • Delicate ring forms & multiple per cell
    • *Crescent shaped gametocyte aftger 7-10 days
  5. P. ovale
    • Enlarged RBC's
    • Oval shaped RBC's
    • Schuffner's dots (eosinophilic stippling)
    • Gametocytes seen 4-18 days after
  6. Toxoplasma gondii
    • typically mild ( mono like symptoms)
    • Can invade tissue in immunocomp.
    • Dx: rising titers of ab's in serum
  7. Babesia species life cycle

    ⇨ B. microti - US
    ⇨ B. divergens - Europe
    Worldwide
    Tick intorduces Sporoozytes

    • Trophozoites develop into merozoites
    • Gametocytes taken back by tick.
  8. Babesia species
    Maltese cross merozoites in RBC's
  9. Trypanasomes -

    ⇨T. cruzi
    • Causes Chagas disease (zoonitic disease)
    • US & Argentina & Poor rural areas of South America
  10. trypanasomes cruzi Life Cycle
    • Insect bites human
    • Human scratches bite pushing in trapanosomes which penetrate various cells.
    • Trapanosomes transform into  amastagote form.
    • Multiply in muscle and tissues
    • Burst out & Enters blood.
  11. T. cruzi clinical disease
    • lesion appears
    • Acute phase typically asymptomatic 
    • Chronic Stage may occur years or decades later

    • Dx: Serology or
    • motile parasites (S or C shaped)
    • Amastagote in muscle
  12. Trypanosoma: African sleeping sickness

    ⇨rhodesiense - East - wild game
    ⇨gambiense - West - human
    Tsetse fly is host

    parasite multiply in gut, transform into epimastigotes than into trypanomatigote
  13. T. brucei disease
    • lesion at bite site
    • mono type symptoms, joint pain, CNS impairment
    • Coma/Death
  14. T. brucei Diagnosis
    • Examine fluid from bite site for trypomastigotes
    • * Late infection use CSF or lymph nodes
  15. Leishmania spp. Life Cycle

    Tropics and subtropics
    • Female Sand Flies - vector borne -
    • Injects promastigotes
    • Promastigotes phagocytized by macrophages and transform into amastigotes.
    • Amastigotes affect tissue & blood
    • Caused by obligate intracellular protozoa
  16. Leishmania forms
    • promastigote: 15-30 μm by 1.5- 4.0 μm
    • Kinetoplast located in the anterior end, where the flagellum leaves the cell
    • Amastigote: inhabits the mammalian host
    • Round tooval
    • clear nucleaus and Central karyosome
    • No free flagellum
  17. leishmania disease
    • lesion with raised edges and central crater.
    • Cutaneous or visceral (kala-azar)

    • Fever, wt loss, swollen glans, spleen & liver.
    • low wbc,rbc,plt count
  18. leishmania dx
    Micropsocpy form bone marrow, spleen, or lymph nodes

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