Ch20 Quiz

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Ch20 Quiz
2013-12-01 09:11:19
Dental Pulp

Ch 20 Quiz
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  1. Estimated HIV infected world wide?
    31-35 million

    2.3-2.8 became infected in 2009
  2. HIV induced oral lesions inĀ adults?
    • Candidiasis (erythematous/pseudomembraneous)
    • Hairy leukoplakia
    • Kaposi sarcoma
    • non-hodgkin's lymphoma
    • periodontal disease (NUG/NUP)

    risk in inverse to CD4 count
  3. Name the 8 identified Herpes viruses?
    • HSV-1
    • HSV-2
    • VZV
    • EBV
    • CMV
    • HH6
    • HH7
    • HH8
  4. Pathophysiology of HSV-1?
    Primary infection: virus transported via retrograde axonal transport to regional sensory ganglia (V), where latency begins

    can be shed in saliva
  5. Clinically aparent recurrent infections are estimated to occur in __% to __% of indviduals harboring latent HSV-1.
  6. Triggers to HSV?
    • Sunlight
    • Trauma
    • Menses
    • Fever
    • Immunosuppression
    • Irritation by dental instruments
  7. Three clinical forms of recurrent HSV-1?
    • 1. Herpes labialis
    • 2. Intraoral recurrence (gingiva or hardpalate)
    • 3. Recurrence mimicking a primary infection
  8. HSV-1 proposed as a potential etiologic factor of pulpal necrosis
  9. VZV establishes latency where?
    Neuronal ganglia (cranial nerve, dorsal root, and autonomic ganglia)
  10. An estimated 13% of zoster cases involve the head and neck.
    may lead to pulpal death
  11. Taurodontism prevalence

    failure of epithelial root sheath differentiation

    most cases molar
  12. How does taurodontism complicate endo?
    ID of canal can be compromised

    Affected teeth more prone to manipulative fracture
  13. Three types of Dens in dente?
    • 1. Enamel lined
    • 2. Enamel lined blind sac that invades the root
    • 3. Invades the root and has secondary foramen
  14. Prevalence of dens-in-dente?

    involves maxillary lateral incisors
  15. Clinical significance of densindente?
    • 1. inc risk of bacteria induced necrosis
    • 2. NRSCT difficult due to complexity

    use CaOH, ultrasonics, GP warm vertical
  16. Den evaginatus prevalence?
    Asian 15%

    premolar and molar
  17. Clinical significance of dens evaginatus?
    narrow shelf of enamel, easily penetrated
  18. Types of DI?
    DI Type 1: assoc with osteogen imperfecta, type 1 collagen defect

    DI Type II/III: DSPP gene defect

    Type II most common 1:6000-8000
  19. Clinical findings of DI?
    Amber-brown to blue-gray hue, cracking or loss of enamel, attrition.

    Radio Type I/II: bulbous crown structure, cervical constriction, short roots, obliterated pulp chamber

    Radio Type III: pulp chamber normal or enlarged, PARLs present
  20. Paget disease of bone (PDB) is a...
    heterogenous, focal, progressive bone disease characterized by active bone turnover

    men over 40 (1%)
  21. Affected sites for PDb?
    pelvis, skull, vertebra, femur, and tibia
  22. Affected bones of PDB manifest an initial ______ phase, followed by a mixed __________/__________, which progresses to a disorganized __________ phase


  23. Clinical signs of PDB?
    osseous distortion or expansion, mild to moderate depp aching bone pain
  24. Skull involvement?

    vestublar involvment in 89%
  25. Dental abnormalities of PDB?
    Malocclusion, hypercementosis, tooth mobility, root resorption, pulp calcification, osteomyelitis, poor fitting prsthesis,
  26. PDB is cotton wool appearance
  27. PDB radiographic findings?
    Hypercementosis, local thickened PDL space, root resorption, pulpal obliteration

    Hyperparathyroidism is generalized widening PDL space
  28. Most prevalent form of rickets is caused by mutations affecting the PHEX gene, termed X-linked hypophosphatemia.
  29. XLH dental findings?
    • Enlarged pulp chamber and pulp horns
    • hypocalcified dentin
    • short roots
    • taurodontism
    • poor defined lamina dura
    • hypoplastic alveolar ridge
  30. What is pathognomonic for XLH?
    Promininent pulp horns extending up to the dentinoenamel junction

    causes spontaneous dental abscesses
  31. Most serious type of thalassemia?
    Beta, known as Cooley anemia
  32. A characteristic "rodent" orĀ "chipmunk" facies may be observed (maxillary protusion or expansion, bossing of skull, prominent cheek bones)
  33. Percent of AA heterozygous for SCA trait?
  34. Sickle red cell lifespan?
    10-30 days
  35. Percent of patients with head and neck manifestations?
  36. Risk of osteomyelitis of the jaws in SCA?
    200 times