Endocrine 5.txt

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Anonymous
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259776
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Endocrine 5.txt
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2014-02-02 11:33:37
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Endocrine
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Endocrine 5
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  1. Depression
    Addison’s
  2. Hypoglycemia
    Addison’s
  3. Postural hypotension
    Addison’s
  4. Weight loss, anorexia
    Addison’s
  5. Bronze skin
    Addison’s
  6. Tachycardia
    Addison’s
  7. GI disturbance
    Addison’s
  8. Weakness, fatigue
    Addison’s
  9. Profound fatigue, dehydration, decrease in BP, decrease Na and increase Ca and K
    Addison’s
  10. Salt craving, hypovolemia
    Addison’s
  11. Hypervolemia
    Cushings
  12. Hyperglycemia
    Cushings
  13. CNS irritability
    Cushings
  14. NA and Fluid retention (edema)
    Cushings
  15. Thin extremities
    Cushings
  16. GI distress increase in acidity
    Cushings
  17. Amenorrhea, hirsutism
    Cushings
  18. Thin Skin
    Cushings
  19. Purple striae
    Cushings
  20. Bruises and petechiae
    Cushings
  21. Osteoporosis
    Cushings
  22. Fat on face and back of shoulders
    Cushings
  23. Gynecomastia
    Cushings
  24. Increase risk of infection
    Cushings
  25. Moon face
    Cushings
  26. Personality changes
    Cushings
  27. Addison’s disease results from
    damage to the adrenal cortex
  28. Symptoms can start from exogenous therapy only lasts 2-4 weeks so don’t want to give a lot of corticosteroids
  29. Causes of addison’s
    Loss of aldosterone and cortisol ages between 30-50
  30. Adrenal cortex secretes
    glucocorticoids (food into energy), immune system inflammatory response, helps body respond to stress; mineralocorticoids (balance of Na, K and H20); androgens
  31. Reduced aldosterone secretion causes
    hyperkalemia, hyponatremia and hypovolemia
  32. Low adrenal androgens
    decreased armpit hair and public
  33. Common cause of secondary adrenal insufficiency
    sudden cessation of long term glucocorticoid therapy; need to be withdrawn gradually
  34. Patients with primary insufficiency
    increased plasma ACTH and melanocyte stimulating hormone
  35. With addison’s stimulation test
    plasma cortisol levels do not rise
  36. FIRST YELLOW Most definitive test for adrenal insufficiency
    ACTH stimulation
  37. Interventions for addison’s
    promote fluid balance, monitor for fluid deficit and prevent hypoglycemia
  38. What corrects glucocorticoid deficiency?
    Hydrocortisone
  39. Addisonian crisis
    Low BP, low BS and high K; cyanosis, fever, s/s of shock, pallor, apprehension, rapid weak pulse, increase RR, confusion, restlessness, loss of consciousness
  40. Hypersecretion of cortisol
    Cushings
  41. Hyperaldosteronism
    excessive mineralcorticoid or androgen production
  42. Pheochromocytoma
    excessive secretion of epi and norepi
  43. Age for Cushing’s
    between 20-50
  44. BP and Cushings’
    increased and headache
  45. Rare that cause of cushing’s
    is from your own body
  46. Cushing’s disease and infection
    high levels of corticosteroids kill lymphocytes; eoisinophils and macrophages are decreased
  47. Moon face and buffalo hump
    Cushing’s
  48. Cushing’s lab tests
    blood, salivary and urine cortisol-usually high; also dexamethasone suppression
  49. Pituitary cushing’s
    ACTH increased
  50. Adrenal cushing’s or chronic steroid use
    ACTH is low
  51. Cushing’s tests
    salivary cortisol (obtained at midnight) and 24 hour urine (obtained for free cortisol and metabolites of cortisol and androgens)
  52. Dexamethasone suppression testing
    cushings
  53. Electrolyte levels in Cushings
    increase Na, decrease Ca and K
  54. Important mgmt. of cushing’s
    handwashing to prevent infection; give glucocorticoids prior to surgery
  55. Surgical mgmt. of adrenalectomy
    assess vitals q 15 and monitor for shoch due to insufficient glucocorticoid replacement
  56. Monitor for GI bleeding
    Cushings
  57. Hypophysectomy
    removal of pituitary; treatment of choice for cushings, stimulation center is gone for thyroid and adrenals
  58. Post hypophysectomy
    don’t brush teeth, cough, sneeze or bend forward

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