Chapter 38

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Author:
3rikita
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273988
Filename:
Chapter 38
Updated:
2014-05-21 23:32:15
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rtt
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  1. what are specific objectives for 0therapy
    correct documented or suspected acute hypoxemia 

    decrease symptoms associated with chronic hypoxemia 

    drecrease workload hypoxemia imposes on the cardiopulmunary system
  2. how does 02 correct hypoxemia
    increases alveolar and blood levels of o2
  3. how does o2 therapy decrease workload on the cardio pulmonary system?
    o2 therapy increases o2 content leading to the heart pumping less blood per minute to meet tissue demands
  4. how does the heart compensate for hypoxemia
    increases ventilation and cardiac output. patients with hypoxemia breathing air can achive acceptable arterial oxygenation only by increasing ventilation this increases the hearts workload..
  5. what lab results can help you determine pt need for o2 therapy
    hemoglobin sat

    PO
  6. what clinical problem or condition can help you determine need for o2 therapy
    post op patients

    co2 or cyanide poisoning 

    shock 

    trauma 

    acute myocardial infarction 

    some premature infants
  7. what are respiratory manifestations on need for o2 therapy?
    • mild > severe
    • tachypnea >tachypnea

    dyspnea > dyspnea 

    paleness > cyanosis
  8. what are cardiovascular manifestations for need of o2 therapy
    mild > severe

    tachycardia > tachycardia, eventually bradycardia, arrhythmia

    mild hypertension, peripheral vasoconstriction > hypertension eventually hypotension
  9. what are neurological manifestations for need of o2 therapy
    mild > severe 

    restlessness > somnolence

    disorientation > confusion

    headaches > distressed appearence

    lassitude > blurred vision

    > tunnel vision

    >loss of coordination

    > impaired judgment

    > slow reaction time

    > manic depression activity

    > coma
  10. what are the effects of enriched o2 environment on the CNS
    in hyperbaric pressure (>1 atm = 760 mm hg)

    • tremors 
    • twitching 
    • convulsions
  11. how can oxigen toxity be avoided?
    • limit patient exposure to 100% to less then 24hrs 
    • high fio2 is ok if concentration is decreased to 70 % with in 2 days and 50%  or less in 5 days
  12. what is the best way to minimize the risk of rop
    keeping arterial po2 in infants less than 80 mm hg 
  13. who is at risk for absorption atelectasis & why
    pt with fio2 greater than .50

    breathing high levels o2 depletes bodys nitrogen
  14. who is at greater risk of absorption atelectasis
    pts breathing at low tidal volumes

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