Cin Path- Pancreas Evaluation.txt

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Author:
Mawad
ID:
289982
Filename:
Cin Path- Pancreas Evaluation.txt
Updated:
2014-11-24 19:59:45
Tags:
clin path pathology
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vetmed
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  1. Blood glucose is controlled primarily by __________.
    endocrine pancreas (glucagon and insulin)
  2. Causes of hypoglycemia. (5)
    pseudohypoglycemia (delayed analysis of sample), insulinoma (increased insulin), hepatic insufficiency (decreased gluconeogenesis), increased utilization (prolonged exercise), sepsis/endotoxemia
  3. Physiologic causes of hyperglycemia. (2)
    postprandial, excitement/fear
  4. Pathologic causes of hyperglycemia. (7)
    diabetes mellitus, pancreatitis, milk fever, hyperadrenocorticism (Cushing's disease), drugs, pancreatic disease, extrapancreatic neoplasia
  5. Digestive disorder resulting from inadequate pancreatic enzyme production, causing malassimilation.
    exocrine pancreas insufficiency (EPI)
  6. Activation of pancreatic digestive enzymes, leading to tissue necrosis, vascular compromise, thrombosis, and edema.
    pancreatitis
  7. α-amylase hydrolyzes ___________, and lipase hydrolyzes _________.
    α-1,4- glycoside linkages; glycerol esters of long chain fatty acids
  8. Amylase and lipase are both cleared primarily by the ________.
    kidney
  9. Elevations of amylase/lipase that are greater than threefold should be attributed to _______________.
    pancreatic damage
  10. Elevations of amylase/lipase that are not greater than threefold can be attributed to... (3)
    decreased GFR, intestinal disease, or hepatic disease
  11. The only source of TLI is the _________.
    pancreas
  12. TLI (trypsin-like immunoreactivity) is increased with ______________.
    pancreatitis (pancreatic cell damage or decreased GFR)
  13. TLI is decreased with _____________.
    exocrine pancreas insufficiency
  14. Negative pancreatic lipase immunoreactivity strongly rules out ____________.
    pancreatitis

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