inflammation and repair NBEO.txt

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Author:
cjensen20
ID:
297057
Filename:
inflammation and repair NBEO.txt
Updated:
2015-02-27 19:24:12
Tags:
systemic inflammation repair NBEO optometry
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systemic inflammation and repair NBEO optometry
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  1. How fast after injury does acute inflammation occur?
    Within 1-2 minutes
  2. What is the role of leukocytes in inflammation and repair?
    • They help clear the injury site of invading bacteria
    • Degrade necrotic by-products
  3. What is the process of acute inflammation?
    • Rubor
    • Calor
    • Dolor
    • Tumor
    • Presence of WBCs
  4. What are the possible outcomes of acute inflammation?
    • Complete resolution
    • Scaring/fibrosis
    • Abcess formation
    • Progression to chronic inflammation
  5. What cells are infiltrated to the site in chronic inflammation:
    • Macrophages
    • Lymphocytes
    • Plasma cells
  6. What 3 processes are occurring simultaneously in chronic inflammation?
    • Active inflammation
    • Tissue injury
    • Healing
  7. Who are the typical culprits of chronic granulomatous bacterial inflammations?
    • TB
    • Leprosy
    • Both mycobacteria
  8. Who are the typical culprits of chronic granulomatous fungal inflammations?
    • Histoplasmosis
    • Blastomycosis
  9. What are the typical culprits of chronic granulomatous foreign body inflammations?
    • Sutures
    • Vascular graft
  10. What are the most common etiologies of binocular, anterior, chronic, granulomatous uveitis?
    TB and Sarcoidosis
  11. What are 7 events that affect wound healing?
    • Local infection
    • Decreased blood supply
    • Decreased peripheral blood flow
    • Immunocompromised states
    • Other systemic infection
    • Malnutrition
    • Increased glucocorticoid production
  12. What reversible causes cellular injury?
    Decreased blood/O2 supply (hypoxia)
  13. What does reversible cell injury do?
    • Causes increase in glycolysis and anaerobic respiration
    • Causes decrease in ATP production
  14. What happens when there is an increase in glycolysis and anaerobic respiration?
    • Increase in lactic acid concentration
    • Decrease in pH
  15. What happens when there is a decrease in ATP production?
    Disrupts the Na/K gradient, leading to edema, and eventual necrosis
  16. What types of necrosis are there?
    • Coagulative
    • Liquefactive
    • Caseous
    • fat
  17. When would you expect to see coagulative necrosis?
    After MI
  18. When would you expect to see liquefactive necrosis?
    Fungal infections in the lung
  19. When would you expect to see caseous necrosis?
    TB infections

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