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lost or limited function of all extremities as a result of damage to spinal cord
lost or limited function in lower extremities and possible trunk
preferred by ASIA; impairment of loss of motor or sensory function in cervical segments of spinal cord
A period of altered reflex activity after traumatic SCI; segments below injury are deprived of input from higher CNS centers.
This is immediately post injury.
Signs & Symptoms:
Flaccid paralysis below injury; absent reflexes & Flaccid bladde
rLasts 1 week to 3 mos., then reflexes resume if reflex arc is intact. So if it is an UMN issue only. If LMN or PNS there won’t be a reflex so no reflex will come back. Increase spasticity in flexor muscles, then after approximately 6-12 mos. is spasticity in extensor muscles
TX very minimal from OTR, no COTA
Compromised status in SCI above T12…Signs and Symptoms: pneumonia, shallow breathing, weak cough, reduced endurance
Abdominals innervated at T7-T12, intercostals at T1-T12, and diaphragm at C4
Complete injuries above C4 need respirator because diaphragm innervates at C4TX: Need deep/diaphragmatic breathing, assisted cough techniques; good body positioning; exercise to strengthen core
An exaggerated response of ANS to nerve irritation/noxious stimuli below level of injury (T6).
Occurs in pts with injury above T6. The ANS will increase BP which cause HR to increase. then the HR drops. eg. Overfull bladder, constipation.
Signs & symptoms: sudden pounding headache, diaphoresis, flushing, goosebumps, chills, anxiety, tachycardia followed by bradycardia
Common causes: blockage in catheter; bowel impaction; bunched up clothing
Tx: Get Help! And find cause quickly and alleviate it; keep pt upright with head elevated to keep BP lower. Remove anything restrictive to reduce BP. Unkink catheter, smooth out clothes.
Life threatening – risk of seizure, stroke, fatality. Don’t leave them alone.
Autonomic dysreflexia=high BP, Low HR