Cardiovascular

Card Set Information

Author:
Omurbek
ID:
306502
Filename:
Cardiovascular
Updated:
2015-08-18 03:16:42
Tags:
Farmacology
Folders:
antiarrhytmic
Description:
antiarrhytmic drugs
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  1. IA names
    Quinidine, Procainamide, Disopyramide
  2. I A MECHANISM
    increase AP duration, increase effective refractory period (ERP) in ventricular action potential, increase QT interval.
  3. 1 A ClINICAl uSE
    Both atrial and ventricular arrhythmias, especially re-entrant and ectopic SVT and VT
  4. 1 A TOXICITY
    Cinchonism (headache, tinnitus with quinidine), reversible SLE-like syndrome (procainamide), heart failure (disopyramide), thrombocytopenia, torsades de pointes due to Џ QT interval.
  5. IB drugs
    Lidocaine, MexileTine. “I’d Buy Liddy’s Mexican Tacos.”
  6. 1 B mechanism
    decrease AP duration. Preferentially affect ischemic or depolarized Purkinje and ventricular tissue. Phenytoin can also fall into the IB category
  7. 1 B clinical use
    Acute ventricular arrhythmias (especially post-MI), digitalis-induced arrhythmias. IB is Best post-MI.
  8. 1 B toxiicity
    CNS stimulation/depression, cardiovascular depression.
  9. 1 C drugs
    Flecainide, Propafenone. “Can I have Fries, Please.”
  10. 1 C mechanism
    Significantly prolongs ERP in AV node and accessory bypass tracts. No effect on ERP in Purkinje and ventricular tissue. Minimal effect on AP duration.
  11. 1 C clinical use
    SVTs, including atrial fibrillation. Only as a last resort in refractory VT.
  12. 1 C clinical use
    SVTs, including atrial 懁brillation. Only as a last resort in refractory VT.
  13. 1 C toxicity
    Proarrhythmic, especially post-MI (contraindicated). IC is Contraindicated in structural and ischemic heart disease.

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