Cattle- Abomasal Dz, Intestinal Dz

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  1. What are the functions of the forestomachs? (3)
    fermentation, particle size, water absorption
  2. What are the functions of the abomasum? (2)
    secreting HCl, secreting digestive juices
  3. What are pre-disposing factors to abomasal displacement? (5)
    GI (abomasal) atony due to change in diet/high conc diet/ketosis, calving
  4. What are the clinical signs of a simple DA? (5)
    decreased milk, decreased appetite, ketosis, ping, splashing
  5. What are the signs of abomasal volvulus? (5)
    all those of simple DA + colic, tachycardia, shock, death
  6. What are the metabolic derangements associated with DA? (4)
    hypochloremia, hypokalemia, metabolic alkalosis, (very late stages) lactic acidosis
  7. Describe the pathogenesis of LDA.
    atony causes abomasum to fill with gas--> becomes buoyant and floats up to left side of rumen--> becomes trapped b/w rumen and left body wall--> partial outflow obstruction
  8. Right DA aka ________.
    right abomasal dilatation
  9. Describe the pathogenesis of RDA.
    atony causes abomasum to float up to right side--> partial outflow obstruction
  10. Describe the pathogenesis of right abomasal volvulus.
    starts as RDA--> twists along a line perpendicular to its long axis--> complete abomasal obstruction--> strangulation of tissue
  11. What are the right flank techniques for correction of a DA? (2)
    omentopexy, abomasopexy
  12. What is the left flank technique for correction of a DA?
  13. With a right flank abomasopexy, the __________ is secured to the body wall.
    pyloric antrum
  14. What are the indications for left flank abomasopexy? (3)
    adhesions, pregnancy, LDA only
  15. With a left flank abomasopexy, suture is placed in the _________ and then passed through the ________ to the ______ of the midline, careful to avoid the _______.
    greater curvature of the abomasum; ventral body wall; right; milk vein
  16. Why is it important to remove sutures two weeks after a left flank abomasopexy?
    it will wick abomasal fluid through the hole and cause a fistula
  17. How do you roll a DA?
    right side down, roll to dorsal recumbancy
  18. What are the types of abomasal ulcers?
    perforating and bleeding ulcers
  19. What factors predispose to abomasal ulcers? (3)
    stress, NSAIDs, lymphosarcoma
  20. What 2 things are protective against abomasal ulcers?
    progesterone of pregnancy, prostaglandin
  21. What are clinical signs of absomasal ulcers? (5)
    off feed, melena, anemia, ileus, abdominal pain
  22. How do you treat abomasal ulcers?
    bleeding: correct metabolic disturbances, reduce stress; perforating: surgery, treat peritonitis, abdominal lavage
  23. Left side pings.
    LDA, rumen gas (usually lower pitched because under less pressure)
  24. Right sided pings that require surgery.
    RDA, RAV, cecal volvulus
  25. Right sided pings that do not require surgery.
    cecal dilation, intestinal gas, uterine gas, abdominal gas
  26. What is really the only disease process that requires a left flank exploratory?
  27. What aspect of surgical treatment of cecal volvulus is very important?
    exteriorization before draining- very contaminated viscous
  28. What is functional intestinal obstruction? What causes it?
    ileus-decreased intestinal motility; mastitis, metritis, toxemia, vagal indigestion
  29. What is anatomic intestinal obstruction? What are types of anatomical obstruction?
    physical obstruction; intraluminal, extraluminal, strangulating, non-strangulating
  30. What type of intestinal obstruction is a surgical emergency?
    anatomic obstruction
  31. What are clinical signs of non-strangulating intestinal obstruction? (7)
    depression, anorexia, decrease GI motility, bloating, low milk, no feces, tenesmus
  32. What are clinical signs of strangulating intestinal obstruction? (5)
    abdominal pain, tachycardia, dehydration, shock, death
  33. What is a consistent finding with all intestinal obstructions?
    dilation of intestines on rectal palpation
  34. How does obstruction cause metabolic alkalosis?
    increased Cl- in rumen, which is more severe the more proximal the obstruction is; K+ follows Cl---> hypokalemia
  35. What will you see on the biochem profile of an animal with intestinal obstruction?
    hypochloremia, hypokalemia, metabolic alkalosis
  36. What is highly suggestive of intestinal obstruction on exploratory?
    distended and non-distended loops of intestine
  37. Invagination of a portion of the intestine into the lumen of adjacent bowel?
  38. The portion of intestines that is inside the adjacent bowel.
  39. The portion of intestine that is receiving the intussusceptum (recipient).
  40. Intussusception tends to occur where...
    the mesentery is longer (jejunojejunal, jejunoileal)
  41. Intussusception is usually ___________ and therefore a surgical emergency.
  42. What are clinical signs of mesenteric root torsion? (6)
    colic, abdominal distension, no feces, distended bowl loops on palpation, tachycardia, dehydration
  43. __________ is a real danger when you correct an obstruction or intestinal volvulus.
Card Set:
Cattle- Abomasal Dz, Intestinal Dz
2015-09-18 05:18:03
vetmed cattle abomasum

vetmed cattle diseases
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