Card Set Information
No known cause-associated risk factors
high sodium intake
excessive calorie intake
coarctation of aorta-narrowing
More musculature -tunica media intima
relay on skeletal muscle to return blood.
stage 2 hypertension
increased renal, peripheral, coronary and cerebral
formation of plaque in artery wall
: Damage to the vessel wall.
: Vessel becomes inflamed.
: Fatty streak appears.
: Plaque develops.
Step 5: Fibrous lesion becomes calcified, hemorrhagic,ulcerated, or thrombosed
S-D pressure 30-40 is normal
dependent rubor and intermittent claudication
decrease risk of PVD
reduce sodium intake
cessation of smoking and limit drinking
surgery to a limb
Caused by nerve damage, loss of sensation, reduced blood flow.
S&S of diabetic ulcers
injury without pain
Arteriole ulcers S&S
Between toes, deeper, inadequate O2 and nutrient supply to deep tissue leading to ulcer.
prolonged venous hypertension, edema leading to stasis ulcers, cellulitis and stasis dermatitis.
Children with PVD
75% in pediatrics
Acute systemic vasculitis
Edema & Erythema
Kawasaki disease treatments in peds
leading cause of heart disease in children-treatment involves-Prevention of coronary artery disease intravenous immunoglobulin is the main treatment second line is corticosteroids
Reyes syndrome in PED
Early S&S-Diarrhea and rapid breathing, confusion, weakness.
causes confusion, brain swelling and liver damage high incidence of occurrence in pediatrics due to ASA Age limit 2 years but children and teenagers shouldn't take ASA.