NUR106 PVD/PED-PVD

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Author:
rmwartenberg
ID:
312284
Filename:
NUR106 PVD/PED-PVD
Updated:
2015-12-20 12:49:24
Tags:
PVD nur106
Folders:
NUR106
Description:
NUR106 PVD
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  1. Primary Hypertension
    • High sodium intake
    • family history
    • excessive calorie intake
    • physical inactivity
  2. Secondary Hypertension
    • renal disease
    • adrenal disease
    • tumors
    • pregnancy 
    • drugs
    • coarctation of aorta-narrowing
  3. Arteries
    are more muscular-tunica intima and media
  4. Venules
    dilate more easily-dependent on skeletal muscle for blood return.
  5. Stage 2 hypertension?
    160/100+
  6. Atherosclerosis
    • formation of plaque within the arteries
    • Step 1: Damage to the vessel wall.
    • Step 2: Vessel becomes inflamed.
    • Step 3:  Fatty streak appears.
    • Step 4:  Plaque develops.
    • Step 5:  Fibrous lesion becomes                 calcified, hemorrhagic, ulcerated, or thrombosed.
  7. Verchows triad
    • venous stasis
    • endothelial damage
    • Hypercoagulability
  8. risk factors for atherosclerosis
    • —Age greater than 60 years old
    • —Family history of HTN
    • —Excessive calorie consumption—
    • Physical inactivity
    • —Excessive alcohol intake—
    • High salt & caffeine intake
    • —Obesity—
    • Smoking—
    • Stress —
    • Decreased intake of K+, Ca++, Mg++
  9. Pulse Pressure
    S-D normal is 30-40mmhg
  10. Arteriole ulcers
    • Deep, usually between toes, blackish intermittent claudication 
    • ulcers created due to a lack of 02 and nutriets. Atherosclerosis predisposes pt. to this.
  11. Venous ulcers aka stasis leg ulcer
    Shallow-weeping wounds that are poor to heal-poor venous return of blood flow.
  12. Types of PVD
    • thrombosis
    • varicose veins
    • stasis dermatitis
    • venous stasis ulcers
  13. Stasis
    • prolonged periods of inactivity
    • HF
    • surgery to a limbg
  14. Endothelial injury
    • tourniquet 
    • IV therapy
  15. Hypercoagulability
    • cancer
    • oral contraceptives
    • dehydration
  16. PVD complications
    • —Venous Insufficiency
    • ◦Prolonged venous hypertension◦
    • Stretch veins◦
    • Damage valves◦
    • Edema◦
    • Waste products build up in tissues
    • Results in different types of diseases-thrombosis, varicose veins, ulcers, stasis dermatitis
  17. Diabetic peripheral disease
    • Nerve damage
    • Loss of sensation
    • reduced blood flow
  18. Diabetic ulcers
    • Uncontrolled blood sugar
    • hyperlipidema 
    • HTN
    • Smoking 
    • Trauma
    • Vitamin deficiencies
    • sedentary life style
    • S&S-neuropathy-inability to recognize objects by feel, injury without pain, paresthesia
  19. PVD for pediatrics
    Same risk factors as adult PVD primary and secondary. and same lifestyle changes to treat PVD
  20. Kawasaki's disease
    75% occur in children
    • Acute systemic vasculitis 
    • Edema
    • erythema
    • conjunctivitis
    • strawberry tongue
    • rash
  21. Reyes syndrome
    • hydrocephalus, liver damage, renal damage
    • Unknown, but higher incidence due to ASA use in pediatrics. Pediatrics should not receive ASA along with teenagers.
  22. Kawasaki treatment
    • Main treatment is prevention of MI/CAD. Primary treatment is Intravenous Immunoglobulin 
    • secondary is corticosteroids
    • ASA may be considered, but will be placed under close observation for reyes

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