Psychiatric Disorders

Home > Flashcards > Print Preview

The flashcards below were created by user bens1088 on FreezingBlue Flashcards. What would you like to do?

  1. Positive
    (presence of abnormal features)
  2. Negative
    (Absence of normal features)
  3. Tardive dyskinesia
    -Old treatment for Schizophrenia

    -Chlorpromazine (sedating). Reserpine (effective but dangerous drop in blood pressure)
  4. Dopamine hypothesis-
    Loss of DA= Parkinsons. Too much DA= schizophrenia

    • -Supported by: Reserpine depletes DA (leak vesciles)
    • -Chlorpromazine blocks DA receptors
    • -Problems: takes weeks to work (cant be all D)
  5. Affective Disorders-
    • -Depression/mania. More sufer from unipolar than bipolar
    • -Amygdala/Prefrontal Cortex/Mesotelencephalic damaged (depression)
  6. Depression/Mania Treatments
    • -Treatment: MAOIs- Prevent breakdown of monoamines
    • -Tricyclic antidepressants- block reuptake of serotonin/norepinephrine (safer then MAOIs)
    • -SSRIs- Very successful
    • -Lithium- lots of side effects
    • -Sleep deprivation improves depression
  7. Monoamine Theory of Depression-
    Underactive serotonin/norepinephrine
  8. Diathesis-Stress model of depression-
    -inherited genetic susception to depression
  9. -HPA axis not as responsive to normal negative feedback
  10. Generalized anxiety disorder-
    stress/anxiety in absence of causal stimulus
  11. Phobia
    - anxiety triggered by stimulus
  12. Panic Disorder
    -onset rapid stress physiological response
  13. OCD
    -anxiety producing obsessive relieved by compulsive actions
  14. PTSD-
    continuing stress following extreme stress
  15. Anxiety Disorder Treatments
    • -Treatments: Benzodiazepines (hypnotics, muscle relaxants) GABA agonist. Increases probability of channel opening
    • -Serotonin agonists- Reduce anxiety without sedation/best long term treatment
  16. Cause of Anxiety?
    • Too little serotonin/GABA neurotransmission=anxiety.
    • Amygdala (fear/defensive maybe responsible)
  17. Barbiturates-
    setative/increased duration of GABA channel opening
  18. Tourettes Syndrome-
    involuntary movement/vocalization

    • -Major genetic component
    • -Hard to study/ no animal models
    • -Treated with neuroleptics
    • -Suggest basal ganglia-thalamus-cortex problem
  19. ADHD Types?
    hyperactive impulsive, inattentive, combined hyperactive impulsive and inattentive
  20. ADHD Brain differences:
    Smaller prefrontal cortex/cerebellum, lower levels of dopamine
  21. ADHD Treatment
    • -Atomoxetine- Norephinephrine reuptakeinhibitor
    • -Amphetamine salts (adderall/retalin) increase dopamine, serotonin and norepinephrine concentrations in synapse

Card Set Information

Psychiatric Disorders
2010-09-09 05:50:46
psych disorders

psych disorder
Show Answers:

What would you like to do?

Home > Flashcards > Print Preview