Hemodynamic monitoring

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Author:
meowzart
ID:
57244
Filename:
Hemodynamic monitoring
Updated:
2010-12-26 17:21:27
Tags:
cvp arterial line blood pressure
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Description:
hemodynamic monitoring in PARR
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  1. A high pressure high resistance system.
    1) systemic
    2) pulmonary
    systemic
  2. A low pressure lowresistance system.1) systemic
    2) pulmonary
    pulmonary
  3. blood pressure =
    flow x resistance
  4. What 3 things determine 'resistance' in the circulatory system?
    • 1) vessel length
    • 2) vessel diameter
    • 3) viscosity of blood
  5. What determines 'flow' of blood pressure equation (flow x resistance = BP)
    cardiac output = HR X sv
  6. What is cardiac output?
    • HR X SV
    • Amount of blood (L/min) pumping through the circulatory system
    • (4-8 l/min)
  7. What 3 components affect cardiac output?
    • 1) preload
    • 2) afterload
    • 3) contractility
  8. What is afterload
    resistance to ventricular ejection
  9. What is preload
    amount of blood volume filling ventricles.
  10. What happens with inadequate preload? (2 things)
    • decreased
    • 1) force of contraction
    • 2) stroke volume
  11. What is contractility?
    ability of myocardial muscle fibres to contract
  12. 5 things that alter preload?
    • 1) total circulating volume
    • 2) venous tone (vasodil or vaso constr)
    • 3) mechanical ventilation/body position
    • 4) ventricular compliance
    • 5) arrhythmias
  13. How does blood volume affect preload?
    • decreased by hypovolemia: diuresis; blood loss.
    • Increased by excess fluids: eg IV solution or heart or renal failure
  14. How does venous tone affect preload?
    • vasodilation decreased preload
    • vasoconstriction increased preload
    • (eg drugs)
  15. How does body position affect preload?
    raise legs increases venous return (by 500 mls)
  16. How does mechanical ventilation affect preload?
    increases intra-thoracic pressure which decreases venous return
  17. how does ventricular compliance affect preload
    non compliant ventricle will elevate preload as the ventricle can't stretch to accomodate
  18. How does arrhythmias affect preload?
    • 30% of stroke volume lost with no p wave. Arrhythmias decrease preload.
    • bradycardia and tachycardia decrease preload.
  19. How can you assess preload? (5 things)
    • a)Heart rate/rhythm
    • b) neck veins
    • c) CVP
    • d) heart sounds (s3/s4 - extra sounds from turbulence of noncompliant ventricles)
    • e) lung sounds/crackles
  20. How does increased afterload affect the patient?
    • increased myocardial oxygen demand
    • (from normal 50% to 90%)
  21. What 4 things can increase left heart afterload?
    • aortic stenosis (impedes flow)
    • increased blood viscosity
    • sympathetic stimulation (vasoconstriction)
    • noncompliant arteries
  22. Does systolic or diastolic BP affect the left ventricular afterload?
    diastolic
  23. What automatic compensatory mechanism occurs when oxygen cannot reach an alveolus?
    capillary surrounding the alveolus constricts
  24. How can you measure right ventricular afterload?
    pulmonary artery catheter. Also by oxygen sats
  25. In COPD or emphysema, is right ventricular afterload increased or decreased
    increased - causes right sided heart failure or corpulmonale
  26. What can affect contractility?
    • 1) drugs (positive inotropes increase; negative inotropes (eg antiarrhythmics) decrease contractility
    • 2)ANS
    • 3) oxygen and ph levels
    • 4) loss of muscle mass
  27. How do drugs affect contractility?
    • Positive inotropes increase contractility (eg; dignoxin, dopamine dobutamine; epi; norepi; phenylephrine)
    • Negative inotropes decrease contractility (eg anti-arrhythmics; lidocaine, procainamide verapamil amiodarone)
  28. Which anti-arrhythmic causes the lease decrease in contractility?
    amiodarone
  29. When using anti-arrhythmics what parameter must be monitored carefully?
    BP - decreasing contractility decreases CO so BP can fall.
  30. How does sympathetic stimulation affect contractility?
    neurotransmitters epi and norepi are released and stimulate beta receptors in heart. this increases heart rate and contractility. (dopamine and dobutamine can mimic this)
  31. How does parasympathetic stimulation affect contractility
    blocks the beta receptors (eg metoprolol and propranolol) decreasing contractility and decreases heart rate

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