pharm exam 2
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pharm exam 2
chapter 8 objectives and review
an additional therapy given to enhance or extend the effect of the primary therapy
substances liberated by the body during phases of inflammation that can produce pain
insensibility to pain without loss of consciousness
agents that relieve pain by inhibitn specific pain pathways
capable of reducing fever
originating or producing within the organism or one of its parts
equal in the ability for giving pain relife
a test to determine risk of bleeding
international normalized ration (INR)
reduction of circulation to an area
an initial high dose to quickly achieve a therapeutic blood level
sensory detection and neuronal transmission of pain stimuli
injuious or harmful
derived from opium a strong dependence producing analgesic
characterist or indicativ (diagnostic) of a particular disease or condition
pain caused by the activation of pain receptors in mucocutaneous and musculoskeletal tissues
pain caused by the activation of pain receptors in internal organs
nonsteroidal anti inflammatory drugs
what is the most common cause to make a pt seek dental services?
All of the following endogenous peptides will relieve
pain except one. Which one is the exception?
what is the most commonly prescribed drug for orofacial pain?
hydrocodone with APAP
when do you use a narcotic versus a nonnarcotic?
depends on the pt pain perception
what is given to people with severe pain?
cox 1 or 3 with oxycodone
what is given to pt with moderate dental pain?
cox 1 inhibitors either alone with APAP or with codeine
t/f APAP can be given to a prego pt
t/f all cox inhibitors increase pain threshold
ASA, ibuprophen, naporoxen and naproxen Na are all examples of what type of inhibitors?
where are cox 1 expressed?
in all normal tissues
t/f cox 1 increase bleeding
t/f cox 1 protects gastric mucosa and causes gastric irritation
true? I dunno jackie those notes seem a little weird but it was in red so i had to put it
where are cox 2 expressed?
primarily in brain, kidneys and female reproduction system and bone
what is the action of cox 2 inhibitors?
raise pain threshold
cox 2 inhibitors block endothelial prostacycline synthesis to cause? 2
where are cox 3 expressed?
what is an example of cox 3
what is an example of cox 2 inhibitor?
t/f cox 3 has no anti inflammatory effect
t/f cox 3 has analgesic and antipyretic effect
true inhibt prostaglandin in CNS
t/f APAP has effect on the platelet function
FALSE NO NO NO effect on platelet function. blah blah blah I am so sick of pharm!
with 500mg of aspirin what happens to bleeding times?
prolongs bleeding times (8 days)
what is the prototype of penicillin?
what antibiotic is prone to resistance (immunity common)?
when do you give premed? 7
aritificial heart valves
specific congenital heart defects
unrepaired or incompletely repaired cyanotic congenital heart disease (shunts and conduits)
completely repaired congenital hear defect with prosthetic material or device during the 1st 6 moths of post surgery
repaired congenital heart defect with residual defect at site or adjacent to site of prostheic patch or device
cardiac transplant that develops valvuar problems
you give premed for a cardiac transplant that has no problems
no only if vavlular problems
t/f premed is needed for cardiac conditions
t/f no pre med is needed for artificial heart valves
false premed needed
what interacts with antibiotics? 3
rifampin (for TB)
if stool appears black while taking antibiotics what should you do?
with what antibiotics do you develop pseudomembranous colitis?
clindamycin and lincomycin