quiz #4- cerebellum II
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quiz #4- cerebellum II
cerebellum- limbic system, hypothalamus
What influences the hypothalamus?
What are the functions of the hypothalamus?
ANS regulation from descending pathways
endocrine regulation from pituitary gland
circadian rythym regulation relays thru hypothalamus to visual systems then project back to pineal gland creating light/dark cycles
limbic system variation
other various things
: temp regulation/feeding
What can disorders of the hypothalamus cause?
temp regulation problems
effect optic/visual system
What does the limbic system control?
emotional, behavioral type of center of the brain
preservation of species (securing food, defense mechanisms, sexual behavior)
Where is the limbic system located?
deep and close to the midline
What are different components of the limbic system?
anterior thalamic nucleus
dorsomedial thalmic nuclei
What is the circuit of papez?
describes structural standpoint connections in the limbic system
starts in hippocampus --> through fornix to mammilary body --> to anterior thalamic nucleus --> to cingulate gyrus and back through cingulum to hippocampus again
Where is the amygdala located?
deep to uncus
rostral to hippocampal formation
near tail of caudate
rostral part of lateral ventricle inf. horn
What is the function of the amygdala?
What did the Kluver-Bucy monkey study reveal?
visual agnosia (wouldn't be scared of snakes)
What can cause Kluver-Bucy syndrome?
chronic alcohol abuse
deep brain injuries
What are other names for thalamic pain syndrome?
central pain syndrome
posterior thalamic pain
What can cause thalamic pain syndrome?
direct injury to thalamus
suprathalamic cortical lesion
primary or secondary tumors
What are signs/symptoms of thalamic pain syndrome?
pain is continous and excrutitating after it starts
: pain elicited by non-noxious stimlui (shaving, make up)
: exaggerated pain response to noxious stimuli
: abnormal pain sensations (dante-esque pain- dante's inferno)
What is central sensitization in thalamic pain syndrome?
: cells associated with pain create it at cortical and thalamic levels
reorganization of mechanoreceptor input
: DC/ML; receptors go to bad places of the thalamus
inappropriate connections w/ nocioceptive pathways
reduced nocioceptive threshold
increased receptive field size causing mulitple neurons to dishcharge with a small stimulation on the skin so the receptive fields overlap
loss of descending inhibitory control over pain pathways in the thalamus, so the ascending pathways go wild