DAH Final

area distinguished by color differentiation from adjacent tissue; flat (freckles)

small circumscribed lesion usually < 1cm in diameter elevated above surface of
normal tissue.

circumscribed elevated lesion > 5mm in diameter with serous fluid, looks like a
blister.

various sized round elevations containing pus

base of a lesion that is flat or broad instead of stem-like.

attached by a stem-like or stalk base (mushroom

feeling of area with fingers. [soft, firm, semifirm, fluid filled

wrinkled tin roof

one compartment or unit that is well defined

ill defined or spread out

erythematous

malignant tumors? (5)

these is a clinical dx which is very common

radiographic dx?

historical dx that occurs in black women, 60's

laboratory dx, elevated serum alkaline phosphatese level

use of biopsy specimen, MAIN component of definitive dx?

rediolucency scalloping around roots
-lesion opened surgically – empty void of bone (fills in)

entrapped salivary gland tissue
developmental anomaly – bilateral
well – circumscribed

aspiration of lesion

bread and butter,
use of above procedures to determine FINAL diagnosis
May use all parts or specific parts
case study

Clusters of ectopic sebaceous glands
lips and buccal mucosa

palatal tori or maxillary tori
occur more commonly in women

usually bilateral

pigment that gives color to skin, eyes, hair, mucosa, and ging

prominent veins located on ventral/lateral surfaces of tongue

sessile nodule on gingival margin of the lingual aspect of mand.
canines

“white line” on buccal mucosa along occlusal plane
-bilateral usually
prominent with bruxism or clenching

generalized opalescence of buccal mucosa (grayish-white)
most commonly in Afro-Americans
disappears when mucosa is stretched

thyroid tissue that has become entrapped in the tissues of the tongue
more common in females – hormonal
located as a mass in midline of dorsal of tongue in the area of the foramen caecum

flat or slightly raised oval or rectangular erythematous area in the midline of dorsal surface of tongue

"benign migratory glossitis"

filiform papillae become elongated and appear white, yellow, black or brown

short duration, arises quickly

movement of WBC's to area of injury

passage of WBC's through endotheial wall of small blood vessels

inflammatory fluid; serum protiens and leukocytes (pus)

abnormal multiplication or increase in the number of normal cells

enlargement of tissue or organ due to an increase in the size of the cell, not the number of cells

temporary increase in # of WBC’s circulating in the blood

or swelling of a lymph node

occurs in the early stage of inflammation; WBC’s occupy the periphery of a blood vessel and adhere to endothelial cells lining the blood vessel

the adherence of WBC’s to the endothelial cells lining an injured blood vessel

containing or forming pus

restoration of damaged or diseased tissue

having a fluid or watery consistency

localized swelling of tissue due to edema, accompanied by sever itching

one area

throughout

mimimal and brief

continued inflammation and longer, wks mos

redness heat swelling pain and loss of nomal tissue function

increase in # of WBC's

plasma fluids & proteins that leave the blood vessels & enter the surrounding tissues.

mainly plasma fluids/protiens thin clear

tissue debris/many WBC's and serous thick/colored

clincal sign of edema(swelling)

a formation of a natural drainage passage boring through tissue allowing
drainage of exudates (Buccal Parulus)

movement of WBC’s to periphery of vessel walls

linng of vessel walls with WBC's

escape of WBC from vessels of plasma fluids that enter tissue

causes of emigration:

also called PMN’s (polymorphonuclear leukocyte)

60-70% of WBC pop.

what are the 3 systems of chemcial mediators

– involved in chronic inflammation & immune response

ingestion of foreign substances by WBC's

become macrophages after emigration

start or enhance the inflammatory response

-Mediates inflammation by causing inc dilation of blood vessels at the site inc permeability of blood vessels by widening gaps between endothelial cells
-Early phase of inflammation

2nd cell to emigrate
aka macrophage

-sequential cascade of plasma proteins that are present in the blood in an activated form
- Release of histamine (mast cells)

clots bld and helps w repair, platelets

fever producing substance

the body’s final defense mechanism in its attempt to restore injured tissue to its original state