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Function of Vascular System
Dilate or constrict depending on metabolic needs of tissues
Oxygenation and waste product removal
Direct the flow of the blood in one continuous direction
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PVD Causes:
Heart failure
thrombus, embolus
atherosclerotic plaque,
chemical or mechanical injury
inflammatory processes
vaso-spastic disorders
congenital malformations
obstruction/pressure
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Number one cause of PVD
plaque build up of the artery , no peripheral pulse that’s a clue of PVD and edema use a doppler
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PVD - Diagnostics
Physical assessment of peripheral pulses
Lab values
Doppler ultrasound flow studies
Duplex ultrasound (imaging of tissues, organs, and vessels)
Exercise tolerance testing (treadmill)
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Plethysmography:
Diagnostic test for PVD
Evaluate arterial flow in lower extremties
Uses a doppler and ultrasound to evaluate flow
May need to wear blood pressure cuffs on your arms and legs for this test.
Compares blood pressure in arms to legs – measuring the ABI (ankle – brachial index
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CT Scan
cross-sectional views of tissues usually with contrast medium
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CT angiography
3-D view of aorta and visceral arteries
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MRI
isolates blood vessels (no contrast medium)
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Arteriosclerosis
hardening of the arteries or full-thickness hardening of small arteries and arterioles
occurs with the aging process, hardening of the arteries
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Atherosclerosis
involves accumulation of lipids, calcium, blood components, carbohydrates, and fibrous tissue on intimal layer of large and medium-sized arteries
the plaque formation
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Arterial Disorders Issue
poor perfusion and oxygenation
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Arterial Disorder Cause
narrowing of the lumen, obstruction by thrombosis, plaque ulceration, aneurysm, rupture
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Arterial Disorder Risk Factors
- diet, HTN, DM, Hyperlipidemia, Stress, smoking,
- sedentary lifestyle, age, family history
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Arterial Disorders Related Illnesses
Hypertension
Angina
Myocardial infarction
Transient ischemic attacks (TIA) and stroke
Aneurysm
Renal vascular disease/end-stage renal disease
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Manifestations of Arterial Disorders
Skin changes r/t color and temperature: “dependent rubor”, or “ elevation pallor”
Diminished or absent pulse
- Ulcers (usually on toes or near toes, +pain, neurologic deficits present
- Dependent on stage or severity of arterial insufficiency
Pain at Rest
Pain or intermittent claudication
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Medical Interventions
Angioplasty and stent placement
Percutaneous Transluminal Angioplasty (PTA)
Laser- Assisted Angioplasty
Atherectomy
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PVD - Treatments (Surgery)
Peripheral arterial bypass operation with autogenous vein to carry blood around the lesion or occlusion.
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Peripheral Arterial bypass Nursing Care
- important to assess for occlusion (pulses, temp etc.)
- report of pain (type of pain)
-bedrest for 18-24 hours
- check blood pressure
- notify surgeon immediately for abnormal findings
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Carotid Endarterectomy (CEA)
opening the carotid artery and removing the obstructing plaque.
Monitor vital signs, neuro status, pulses, bleeding,
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Arterial Disorder Complications from interventions
Hematoma
Embolus
Dissection
Bleeding
Stent embolization or dislodgement
Vessel damage
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Nursing Interventions for Arterial Disorders
Positioning affected part below heart level
If edema, position slightly elevated but not above the heart
Walking or graded isometric exercises to promote circulation and development of collateral circulation
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Patient teaching for Arterial Disorders
- medications (vasodilators, lipid lowering agents,
- anti-platelet agents)
No smoking!
surgery
applications of warmth,
avoidance of cold temperatures,
avoid heating pads and hot-water bottles
avoid emotional upsets,
removal of restrictive clothing or accessories
avoid crossing the legs, inspection of legs and feet
nutrition: weight reduction
Diet low fat, low sodium,
Encourage soy proteins, folic acid and B6 and B12 vitamins
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What benefit does folic acid have on arterial disorders?
- Folic acid on arterial disorders: homosistene when high causes more plaque formation so when you give
- folic acid it decreases it
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What benefit does soy have on arterial disorders?
Soy lowers your cholesterol level
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Peripheral Arterial Occlusive Disease
Legs most effected, men 50 + years
Below renal arteries to popliteal artery
Aching, cramping, fatigue, weakness relieved with rest; with severity, pain worse at night/at rest
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Upper extremity Arterial Occlusive Disease
Effects the arms/upper extremities
Unilateral coolness, discrepancy of 20 mm HG or more on BP right to left
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Thromboangiitis Obliterans or Buerger’s Disease
Autoimmune vasculitis resulting in in microscopic occlusion of distal vessels caused by heavy smoking/chewing tobacco
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Buergers think about
smoking directly related causes vessel to inflame and have narrowing of the vessels
When pt stops smoking inflammation does subside
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Raynaud’s Disease
extreme sensitivity to cold aggravated by nicotine, emotion, chilling, limited to hands and feet
Phenomenon (usually unilaterally)
Treatment and education related to preventing vasoconstriction
Can be stress, cold, nicotine , usually one hand or two fingers and promote vasodilation
Reduce stress, wear gloves
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Aortic Aneurysm
localized sac or dilation (thoracic, abdominal, dissecting aneurysm)
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Arterial Embolism and Arterial Thrombosis
occlusion/clot
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Venous Insufficiency
Results of prolonged venous hypertension that stretches the vein and damages the valves
Damage and hypertension leads to back-up of blood resulting in edema
Difficulty eliminating waste and thus they build-up in the tissues resulting in stasis, ulcers, edema, cellulitis
Usually around ankles
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Venous Disorders
Venous thrombosis
Deep vein thrombosis
Thrombophlebitis
Virchow’s triad:
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Virchow’s triad
VENOUS STASIS, DAMAGE TO INTIMAL LINING, INCREASED BLOOD VISCOSITY
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Who’s at risk for Venous Diosorders
elderly, bed ridden, post-op patients, oral contraceptives, HRT, smokers, pregnant females, obese, people who stand for a prolonged period of time
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Manifestations – Venous Disorders
Pain, warmth, redness, and edema
Prominence of superficial veins
Positive Homan’s sign
Signs of pulmonary embolus
Ulcers (usually ankle area, +pulses, no claudication, no neurologic deficits)
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Therapies
Elastic stockings, pneumonic compression device, positioning, exercise
Heparin IV or SQ
Coumadin oral
Low-molecular weight heparin (Lovenox), less risk of thrombocytopenia
Thrombolytics
ASA, Ecotrin, Bayer, Plavix
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Heparin IV or SQ what labs do you look at
- PTT (partial thromboplastin time), 2-3x average
- INR (international normalized ratio)2.0-3.0
platelet count
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Coumadin Oral Labs to look at
- PT (prothrombin time) 1.5-2x average
- INR 2.0-3.0
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Drug Therapy- Risks
Bleeding
Drug interactions: (ASA, anabolic steroids,, glucagon, chloramphenicol, neomycin, etc) POTENTIATE oral anticoagulant effects
- Drug interactions: (Dilantin, barbituates, diuretics,
- estrogens), DECREASE anticoagulant effects
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Management of Chronic Venous Insufficiency
leg elevation,
elastic stockings
removal of restrictive garments
proper nutrition
Collaborate with Wound care specialist
Intermittent pneumatic compression pump
Varicose veins and venous reflux: VNUS procedure
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